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hepatic ischemia

Known as: liver ischemia 
National Institutes of Health

Related topics

Related topics

1 relation
hepatic infarction

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
Highly Cited
2014
Highly Cited
2014
Melatonin Inhibits mTOR-Dependent Autophagy during Liver Ischemia/Reperfusion
Background: Autophagy is a self-digestion system responsible for maintaining cellular homeostasis and interacts with reactive… 
Highly Cited
2006
Highly Cited
2006
Critical role of acidic sphingomyelinase in murine hepatic ischemia‐reperfusion injury
The molecular mechanisms of hepatic ischemia/reperfusion (I/R) damage are incompletely understood. We investigated the role of… 
Highly Cited
2004
Highly Cited
2004
Role of the permeability transition pore in cytochrome C release from mitochondria during ischemia-reperfusion in rat liver.
Highly Cited
2003
Highly Cited
2003
Regulation of postischemic liver injury following different durations of ischemia.
The objective of this study was to define the relationship among Kupffer cells, O(2)(-) production, and TNF-alpha expression in… 
Highly Cited
2001
Highly Cited
2001
Cytochrome c release into cytosol with subsequent caspase activation during warm ischemia in rat liver.
Apoptosis plays an important role in liver ischemia and reperfusion (I/R) injury. However, the molecular basis of apoptosis in I… 
Highly Cited
1999
Highly Cited
1999
Evaluation of a novel bioartificial liver in rats with complete liver ischemia: treatment efficacy and species-specific alpha-GST detection to monitor hepatocyte viability.
Highly Cited
1995
Highly Cited
1995
Hepatic release of endothelin-1 after warm ischemia. Reperfusion injury and its hemodynamic effect.
This study investigated the release of endothelin (ET)-1 from the liver after warm ischemia/reperfusion (I/R) injury. Wistar rats… 
Highly Cited
1995
Highly Cited
1995
INHIBITION OF NITRIC OXIDE SYNTHESIS AGGRAVATES REPERFUSION INJURY AFTER HEPATIC ISCHEMIA AND ENDOTOXEMIA
The potential role of nitric oxide (NO) was investigated in the pathophysiology of liver injury after priming with 20 min hepatic… 
Highly Cited
1991
Highly Cited
1991
Vascular oxidant stress and hepatic ischemia/reperfusion injury.
The objective of this study was to test the hypothesis that the extracellular oxidation of glutathione (GSH) may represent an… 
Highly Cited
1980
Highly Cited
1980
An experimental study of survival after two hours of normothermic hepatic ischemia.
Total hepatic inflow occlusion is well tolerated in pigs with normothermia for as long as two hours, provided that splanchnic… 
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