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diethylamine dinitric oxide adduct

Known as: 2-(N,N-diethylamino)-diazenolate-2-oxide 
National Institutes of Health

Related topics

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2 relations

Broader (2)

DiethylaminesNitric Oxide Donors

Papers overview

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2017
2017
Enhanced Electrical Field Stimulated Nitrergic and Purinergic Vasoreactivity in Distal vs Proximal Internal Pudendal Arteries.
BACKGROUND The internal pudendal arteries (IPAs) supply blood to the penis and are highly susceptible to vascular remodeling in… Expand
Highly Cited
2009
Highly Cited
2009
Direct Impairment of Vascular Function by Diesel Exhaust Particulate through Reduced Bioavailability of Endothelium-Derived Nitric Oxide Induced by Superoxide Free Radicals
Background Diesel exhaust particulate (DEP) is a key arbiter of the adverse cardiovascular effects of air pollution. Objectives… Expand
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2008
2008
Pharmacological profile of store-operated Ca(2+) entry in intrapulmonary artery smooth muscle cells.
Store-operated Ca(2+) entry (SOCE) plays an important role in the contraction and proliferation of pulmonary artery smooth muscle… Expand
2006
2006
Nitric oxide-evoked glutamate release and cGMP production in cerebellar slices: Control by presynaptic 5-HT1D receptors
We previously reported that pre- and postsynaptic 5-hydroxytryptamine (5-HT) receptors effectively control glutamatergic… Expand
Highly Cited
2003
Highly Cited
2003
NO enhances presynaptic currents during cerebellar mossy fiber-granule cell LTP.
Nitric oxide (NO) is a candidate retrograde messenger in long-term potentiation (LTP). The NO metabolic pathway is expressed in… Expand
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2002
2002
NO donors potentiate the β‐adrenergic stimulation of ICa,L and the muscarinic activation of IK,ACh in rat cardiac myocytes
The effects of nitric oxide (NO) donors on the L‐type Ca2+ current (ICa,L) and the muscarinic activated K+ current (IK,ACh) were… Expand
Highly Cited
2000
Highly Cited
2000
Mechanisms of NO/cGMP-dependent vasorelaxation.
Both cGMP-dependent and -independent mechanisms have been implicated in the regulation of vascular tone by NO. We analyzed… Expand
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2000
2000
Nitric oxide-mediated heme oxidation and selective beta-globin nitrosation of hemoglobin from normal and sickle erythrocytes.
Nitric oxide (NO) has been reported to modulate the oxygen affinity of blood from sickle cell patients (SS), but not that of… Expand
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