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ZFP57 gene

Known as: ZNF698, ZFP57, bA145L22.2 
 
National Institutes of Health

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
2016
2016
Genomic imprinting defect in Zfp57 mutant iPS cell lines.
ZFP57 maintains genomic imprinting in mouse embryos and ES cells. To test its roles during iPS reprogramming,we derived iPS… Expand
Review
2016
Review
2016
Maintaining memory of silencing at imprinted differentially methylated regions
Imprinted genes are an exceptional cluster of genes which are expressed in a parent-of-origin dependent fashion. This allele… Expand
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2015
2015
Maternal and zygotic Zfp57 modulate NOTCH signaling in cardiac development
Significance Abnormal heart development is a common birth defect. Genomic imprinting is absolutely essential for mammalian… Expand
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2014
2014
The stem cell transcription factor ZFP57 induces IGF2 expression to promote anchorage-independent growth in cancer cells
Several common biological properties between cancer cells and embryonic stem (ES) cells suggest the possibility that some genes… Expand
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Review
2013
Review
2013
On how mammalian transcription factors recognize methylated DNA
DNA methylation is an epigenetic mark that is essential for the development of mammals; it is frequently altered in diseases… Expand
Highly Cited
2011
Highly Cited
2011
In Embryonic Stem Cells, ZFP57/KAP1 Recognize a Methylated Hexanucleotide to Affect Chromatin and DNA Methylation of Imprinting Control Regions
Summary The maintenance of H3K9 and DNA methylation at imprinting control regions (ICRs) during early embryogenesis is key to the… Expand
Highly Cited
2011
Highly Cited
2011
Zinc Finger Protein ZFP57 Requires Its Co-factor to Recruit DNA Methyltransferases and Maintains DNA Methylation Imprint in Embryonic Stem Cells via Its Transcriptional Repression Domain*
Background: ZFP57 is a maternal-zygotic effect gene that maintains genomic imprinting in mouse embryos. Results: KAP1 facilitates… Expand
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2009
2009
Screening for genomic variants in ZFP57 in Silver-Russell syndrome patients with 11p15 epimutations.
Silver-Russell syndrome (SRS) describes a uniform malformation syndrome characterized by intrauterine and postnatal growth… Expand
2009
2009
DNA hypomethylation, transient neonatal diabetes, and prune belly sequence in one of two identical twins
One known genetic mechanism for transient neonatal diabetes is loss of methylation at 6q24. The etiology of prune belly sequence… Expand
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