Skip to search formSkip to main contentSkip to account menu

YCG 063

Known as: YCG-063, YCG063 
National Institutes of Health

Related topics

Related topics

2 relations

Broader (2)

HydrazonesNitroimidazoles

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
2019
2019
[Role of Reactive Oxygen Species in GDC-0152-Induced Apoptosis and Autophagy of NB4 cells].
OBJECTIVE To investigate the effect of reactive oxygen species (ROS) on GDC-0152-induced apoptosis and autophagy of acute… 
2015
2015
3,3'-Diindolylmethane inhibits VEGF expression through the HIF-1α and NF-κB pathways in human retinal pigment epithelial cells under chemical hypoxic conditions.
Oxidative stress in the retinal pigment epithelium (RPE) can lead to the pathological causes of age-related macular degeneration… 
2015
2015
YCG063 inhibits Pseudomonas aeruginosa LPS-induced inflammation in human retinal pigment epithelial cells through the TLR2-mediated AKT/NF-κB pathway and ROS-independent pathways.
YCG063 is known as an inhibitor of reactive oxygen species (ROS); however, its intracellular mechanisms of action remain poorly… 
2014
2014
Tacrine induces apoptosis through lysosome- and mitochondria-dependent pathway in HepG2 cells.
2013
2013
Overexpression of Smad7 suppressed ROS/MMP9-dependent collagen synthesis through regulation of heme oxygenase-1
We previously reported that AngiotensinII receptor blocker effectively inhibited TGF-β1-mediated epithelial-to-mesenchymal… 
By clicking accept or continuing to use the site, you agree to the terms outlined in our Privacy Policy (opens in a new tab), Terms of Service (opens in a new tab), and Dataset License (opens in a new tab)