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Xeroderma pigmentosum, group G

Known as: XERODERMA PIGMENTOSUM VII, XP, GROUP G, XPGC 
 
National Institutes of Health

Papers overview

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Review
2013
Review
2013
SIGNIFICANCE Oxidative DNA damage is repaired by multiple, overlapping DNA repair pathways. Accumulating evidence supports the… Expand
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Highly Cited
2007
Highly Cited
2007
Mutations in the human XPG gene give rise to an inherited photosensitive disorder, xeroderma pigmentosum (XP) associated with… Expand
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Highly Cited
2005
Highly Cited
2005
Loss of a nonenzymatic function of XPG results in defective transcription-coupled repair (TCR), Cockayne syndrome (CS), and early… Expand
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Highly Cited
2004
Highly Cited
2004
Xeroderma pigmentosum (XP) is a human genetic disease which is caused by defects in nucleotide excision repair. Since this repair… Expand
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Review
1999
Review
1999
From its very beginning, life has faced the fundamental problem that the form in which genetic information is stored is not… Expand
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Highly Cited
1997
Highly Cited
1997
Nucleotide excision repair in humans is a complex reaction involving 14 polypeptides in six repair factors for dual incisions on… Expand
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Highly Cited
1997
Highly Cited
1997
During nucleotide excision repair in human cells, a damaged DNA strand is cleaved by two endonucleases, XPG on the 3' side of the… Expand
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Highly Cited
1995
Highly Cited
1995
Nucleotide excision repair is the principal way by which human cells remove UV damage from DNA. Human cell extracts were… Expand
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Highly Cited
1995
Highly Cited
1995
Human DNA repair excision nuclease removes DNA damage by incising on both sides of the lesion in a precise manner. The activity… Expand
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Highly Cited
1994
Highly Cited
1994
HUMANS with a defect in the XPG protein suffer from xeroderma pigmentosum (XP) resulting from an inability to perform DNA… Expand
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