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VAS 2870

Known as: VAS-2870, VAS2870 
National Institutes of Health

Papers overview

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2020
2020
Differential Effects of Reactive Oxygen Species on IgG versus IgM Levels in TLR-Stimulated B Cells
Key Points TLR stimulation of B cells into Ig-secreting plasma cells requires NOX-dependent ROS. The high secretion of IgG per… 
2017
2017
Nox2 contributes to hyperinsulinemia-induced redox imbalance and impaired vascular function
Review
2017
Review
2017
NADPH oxidase: its potential role in promotion of pulmonary arterial hypertension
NADPH oxidases (NOXs) are a group of enzymes for superoxide anion (O2·−) generation through transferring electrons from NADPH to… 
2016
2016
Nox4 Plays a Role in TGF-β–Dependent Lens Epithelial to Mesenchymal Transition
Purpose Transforming growth factor-β induces an epithelial to mesenchymal transition (EMT) in the lens, presented as an aberrant… 
2016
2016
NADPH oxidase inhibitor regulates microRNAs with improved outcome after mechanical reperfusion
Background Inhibition of the nicotinamide adenine dinucleotide phosphate (NADPH) oxidase (NOX) pathway improves the neurological… 
2016
2016
Mechanical strain stimulates vasculogenesis and expression of angiogenesis guidance molecules of embryonic stem cells through elevation of intracellular calcium, reactive oxygen species and nitric…
Highly Cited
2013
Highly Cited
2013
NADPH oxidase-dependent and -independent mechanisms of reported inhibitors of reactive oxygen generation
NADPH oxidase isoform-2 (NOX2) generates reactive oxygen species (ROS) that contribute to neurodegenerative and cardiovascular… 
2013
2013
Selective inhibition of NADPH oxidase reverses the over contraction of diabetic rat aorta☆
Highly Cited
2012
Highly Cited
2012
H2O2 Production Downstream of FLT3 Is Mediated by p22phox in the Endoplasmic Reticulum and Is Required for STAT5 Signalling
The internal tandem duplication (ITD) of the juxtamembrane region of the FLT3 receptor has been associated with increased… 
2012
2012
Activation of PPARδ prevents endothelial dysfunction induced by overexpression of amyloid-β precursor protein.
AIMS Existing evidence suggests that amyloid-β precursor protein (APP) causes endothelial dysfunction and contributes to… 
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