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SIGMAR1 gene

Known as: SIGMA NONOPIOID INTRACELLULAR RECEPTOR 1, sigma non-opioid intracellular receptor 1, SIGMAR1 
National Institutes of Health

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
2018
2018
Determining Whether Maternal Fetal Triage Index Implementation Decreases Patient Walk‐Out Rates
2017
2017
GNE missense mutation in recessive familial amyotrophic lateral sclerosis
Amyotrophic lateral sclerosis (ALS) is a motor neuron disease eventually leading to death from respiratory failure. Recessive… 
2016
2016
Abstract 222: Sigmar1 Mediates Mitochondrial Autophagy and Protects the Heart Against Ischemia/Reperfusion Injury
Rationale: Sigma 1 receptor (Sigmar1) is a highly expressed mitochondrion-associated ER membrane resident protein in different… 
2016
2016
Abstract 18563: Sigmar1 Protects the Heart Against Ischemia/Reperfusion Injury
Introduction: Accumulative evidence indicates that increased expression and presumably activity of molecular chaperones are… 
2015
2015
Is SIGMAR1 a confirmed FTD/MND gene?
Dear Sir, Frontotemporal dementia (FTD) and the related condition motor neuron disease (MND) can have a genetic component to… 
2015
2015
A Study of Island Size and Bit Aspect Ratio Factors in Staggered BPMR Channel with SRBP Method
2010
2010
The synthesis, structure, and properties of group 2 poly(pyrazolyl)borates and their use for the atomic layer deposition of group 2 borates
THE SYNTHESIS, STRUCTURE, AND PROPERTIES OF GROUP 2 POLY(PYRAZOLYL)BORATES AND THEIR USE FOR THE ATOMIC LAYER DEPOSITION OF GROUP… 
2004
2004
Lack of association between sigma1 receptor gene variants and schizophrenia
Abstract  Several pharmacological studies suggest the possible involvement of sigma1 receptors in the pathogenesis of… 
Highly Cited
2002
Highly Cited
2002
Analysis of sigma receptor (sigmaR1) expression in retinal ganglion cells cultured under hyperglycemic conditions and in diabetic mice.
Highly Cited
2001
Highly Cited
2001
Expression pattern of the type 1 sigma receptor in the brain and identity of critical anionic amino acid residues in the ligand-binding domain of the receptor.
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