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RU-28362

Known as: RU 28362 
National Institutes of Health

Papers overview

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1995
1995
[3H]-aldosterone binding sites (type I receptors) in the lateral wall of the cochlea: distribution assessment by quantitative autoradiography.
Mineralocorticoids, such as aldosterone, are steroids that enhance Na+ retention and K+ excretion in ion-transporting epithelial… 
1994
1994
Comparison of aldosterone- and RU-28362-induced apical Na+ and K+ conductances in rat distal colon.
In mammalian distal colon, aldosterone induces electrogenic Na+ absorption and electrogenic K+ secretion, whereas the sole… 
1994
1994
In contrast to glucocorticoid receptors, mineralocorticoid receptors are not autoregulated in rat distal colon epithelia.
Glucocorticoids and mineralocorticoids exert profound effects on electrolyte balance in the rat distal colon by binding with high… 
1994
1994
Intracranial Dehydroepiandrosterone Blocks the Activation of Tryptophan Hydroxylase in Response to Acute Sound Stress
Bilateral infusion of dehydroepiandrosterone (DHEA) given intracerebroventricularly blocked the sound stress-induced increase in… 
1993
1993
Corticosterone is a preferable ligand for measuring rat brain corticosteroid receptors: competition by RU 28362 and RU 26752 for dexamethasone binding in rat hippocampal cytosol
1992
1992
Footshock stress-induced supersensitivity to isoprenaline in the isolated pacemaker of the rat: effects of the compounds RU-38486 and RU-28362.
1990
1990
Analysis of the glucocorticoid receptor during differentiation of 3T3-F442A preadipocyte cell line in culture.
1989
1989
Receptor specificity of a glucocorticoid- and stress-induced hippocampal protein
Following treatment of rats with a subcutaneous injection of 5 mg of corticosterone, hippocampal slices in vitro show increased… 
1988
1988
Glucocorticoids have a different action than aldosterone on target tissue.
Studies were performed, using a synthetic glucocorticoid, RU 28362, that has negligible binding to aldosterone receptors, to… 
1987
1987
Glycyrrhizic acid and its hydrolysate as mineralocorticoid agonist.
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