Skip to search form
Skip to main content
Skip to account menu
Semantic Scholar
Semantic Scholar's Logo
Search 209,921,006 papers from all fields of science
Search
Sign In
Create Free Account
Protease-activated Receptor-1 Antagonist [EPC]
Known as:
PAR-1 Antagonist
, Protease-activated Receptor-1 (PAR-1) Antagonist
, Protease-activated Receptor-1 Antagonist
National Institutes of Health
Create Alert
Alert
Papers overview
Semantic Scholar uses AI to extract papers important to this topic.
2019
2019
High-endothelial cell-derived S1P regulates dendritic cell localization and vascular integrity in the lymph node
S. Simmons
,
N. Sasaki
,
+13 authors
M. Ishii
eLife
2019
Corpus ID: 203625711
While the sphingosine-1-phosphate (S1P)/sphingosine-1-phosphate receptor-1 (S1PR1) axis is critically important for lymphocyte…
Expand
2015
2015
An Allelic Series of bak1 Mutations Differentially Alter bir1 Cell Death, Immune Response, Growth, and Root Development Phenotypes in Arabidopsis thaliana
Michael P Wierzba
,
F. Tax
Genetics
2015
Corpus ID: 34757082
Receptor-like kinases (RLKs) mediate cell-signaling pathways in Arabidopsis thaliana, including those controlling growth and…
Expand
2013
2013
Prokineticin Receptor‐1 Is a New Regulator of Endothelial Insulin Uptake and Capillary Formation to Control Insulin Sensitivity and Cardiovascular and Kidney Functions
Mojdeh Dormishian
,
Gulen Turkeri
,
+6 authors
C. Nebigil
Journal of the American Heart Association…
2013
Corpus ID: 11588704
Background Reciprocal relationships between endothelial dysfunction and insulin resistance result in a vicious cycle of…
Expand
2012
2012
Amid derivatives of n-carbamide-substituted amino acids as modulators of formylpeptide receptor-1 (fprl-1)
Ричард Л. Бирд
,
Тьен Т. Дуонг
,
Джон Е. Донелло
,
Веена Висванат
,
Майкл Е. Гарст
2012
Corpus ID: 104193764
FIELD: chemistry. SUBSTANCE: invention relates to a compoundd represented by the formula , its enantiomers, diastereoisomers or…
Expand
2011
2011
Genetic Inactivation of Prokineticin Receptor-1 Leads to Heart and Kidney Disorders
Mounia Boulberdaa
,
Gulen Turkeri
,
+7 authors
C. Nebigil
Arteriosclerosis, Thrombosis and Vascular Biology
2011
Corpus ID: 14758416
Objective—Prokineticins are potent angiogenic hormones that use 2 receptors, prokineticin receptor-1 (PKR1) and PKR2, with…
Expand
Highly Cited
2008
Highly Cited
2008
Prokineticin Receptor-1 Induces Neovascularization and Epicardial-Derived Progenitor Cell Differentiation
K. Urayama
,
C. Guilini
,
+5 authors
C. Nebigil
Arteriosclerosis, Thrombosis and Vascular Biology
2008
Corpus ID: 6834270
Objective—Identification of novel factors that contribute to myocardial repair and collateral vessel growth hold promise for…
Expand
Highly Cited
2007
Highly Cited
2007
The prokineticin receptor‐1 (GPR73) promotes cardiomyocyte survival and angiogenesis
K. Urayama
,
C. Guilini
,
+5 authors
C. Nebigil
The FASEB Journal
2007
Corpus ID: 9013802
Prokineticins are potent angiogenic factors that bind to two G protein‐coupled receptors to initiate their biological effects. We…
Expand
Highly Cited
2007
Highly Cited
2007
Molecular dissection of the myelin-associated glycoprotein receptor complex reveals cell type–specific mechanisms for neurite outgrowth inhibition
K. Venkatesh
,
Onanong Chivatakarn
,
S. Sheu
,
R. Giger
Journal of Cell Biology
2007
Corpus ID: 18648805
Neuronal Nogo66 receptor-1 (NgR1) binds the myelin inhibitors NogoA, OMgp, and myelin-associated glycoprotein (MAG) and has been…
Expand
Highly Cited
2003
Highly Cited
2003
Chemical Coding of GABAB Receptor‐Immunoreactive Neurones in Hypothalamic Regions Regulating Body Weight
M. Bäckberg
,
M. Collin
,
M. Ovesjö
,
B. Meister
Journal of neuroendocrinology
2003
Corpus ID: 44615263
γ‐aminobutyric acid (GABA) interacts with hypothalamic neuronal pathways regulating feeding behaviour. GABA has been reported to…
Expand
By clicking accept or continuing to use the site, you agree to the terms outlined in our
Privacy Policy
(opens in a new tab)
,
Terms of Service
(opens in a new tab)
, and
Dataset License
(opens in a new tab)
ACCEPT & CONTINUE