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PD 166793

Known as: PD-166793, PD166793 
 
National Institutes of Health

Papers overview

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2012
2012
Loss of Timp3 gene leads to abdominal aortic aneurysm formation in response to angiotensin II.
Aortic aneurysm is dilation of the aorta primarily due to degradation of the aortic wall extracellular matrix (ECM). Tissue… Expand
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Highly Cited
2007
Highly Cited
2007
Matrix metalloproteinase-2 degrades the cytoskeletal protein alpha-actinin in peroxynitrite mediated myocardial injury.
Matrix metalloproteinase (MMP)-2 mediates myocardial ischemia-reperfusion injury which is characterized by enhanced peroxynitrite… Expand
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Highly Cited
2005
Highly Cited
2005
Expression of MMP2, MMP9 and MMP3 in Breast Cancer Brain Metastasis in a Rat Model
In order to study the expression of MMP2, MMP3 and MMP9 in breast cancer brain metastasis, we used a syngeneic rat model of… Expand
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Highly Cited
2005
Highly Cited
2005
Accelerated LV remodeling after myocardial infarction in TIMP-1-deficient mice: effects of exogenous MMP inhibition.
Alterations in matrix metalloproteinases (MMPs) and tissue inhibitors of MMPs (TIMPs) have been implicated in adverse left… Expand
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Highly Cited
2005
Highly Cited
2005
Combination of tumor necrosis factor-alpha ablation and matrix metalloproteinase inhibition prevents heart failure after pressure overload in tissue inhibitor of metalloproteinase-3 knock-out mice.
Cytokine and extracellular matrix (ECM) homeostasis are distinct systems that are each dysregulated in heart failure. Here we… Expand
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2003
2003
Matrix metalloproteinase inhibition modifies left ventricular remodeling after myocardial infarction in pigs.
BACKGROUND Global and regional shape changes that occur within the left ventricular wall after myocardial infarction have been… Expand
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Highly Cited
2003
Highly Cited
2003
Myocardial infarct expansion and matrix metalloproteinase inhibition.
BACKGROUND A potential mechanism for left ventricular (LV) remodeling after myocardial infarction (MI) is activation of the… Expand
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Highly Cited
2002
Highly Cited
2002
Peroxynitrite-induced myocardial injury is mediated through matrix metalloproteinase-2.
OBJECTIVES Peroxynitrite (ONOO(-)) mediates in part both ischemia-reperfusion and pro-inflammatory cytokine-induced injury to the… Expand
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Highly Cited
2001
Highly Cited
2001
Matrix metalloproteinase inhibition attenuates left ventricular remodeling and dysfunction in a rat model of progressive heart failure.
BACKGROUND Matrix metalloproteinase (MMP) activation contributes to tissue remodeling in several disease states, and increased… Expand
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Highly Cited
1999
Highly Cited
1999
Matrix metalloproteinase inhibition during the development of congestive heart failure : effects on left ventricular dimensions and function.
The development of congestive heart failure (CHF) is associated with left ventricle (LV) dilation and myocardial remodeling. The… Expand
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