PD 166793

Known as: PD-166793, PD166793 
 
National Institutes of Health

Topic mentions per year

Topic mentions per year

1999-2016
02419992016

Papers overview

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2012
2012
Aortic aneurysm is dilation of the aorta primarily due to degradation of the aortic wall extracellular matrix (ECM). Tissue… (More)
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Highly Cited
2005
Highly Cited
2005
In order to study the expression of MMP2, MMP3 and MMP9 in breast cancer brain metastasis, we used a syngeneic rat model of… (More)
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2005
2005
To assess the molecular changes associated with pancreatic beta-cell dysfunction occurring during the onset of type 2 diabetes… (More)
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Highly Cited
2003
Highly Cited
2003
BACKGROUND A potential mechanism for left ventricular (LV) remodeling after myocardial infarction (MI) is activation of the… (More)
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2003
2003
BACKGROUND Global and regional shape changes that occur within the left ventricular wall after myocardial infarction have been… (More)
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2003
2003
BACKGROUND A cause-effect relationship has been established between matrix metalloproteinases (MMPs) and left ventricular (LV… (More)
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Highly Cited
2002
Highly Cited
2002
OBJECTIVES Peroxynitrite (ONOO(-)) mediates in part both ischemia-reperfusion and pro-inflammatory cytokine-induced injury to the… (More)
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Highly Cited
2001
Highly Cited
2001
BACKGROUND Matrix metalloproteinase (MMP) activation contributes to tissue remodeling in several disease states, and increased… (More)
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Highly Cited
2000
Highly Cited
2000
OBJECTIVE Determine the effect of a matrix metalloproteinase inhibitor (MMPi) and angiotensin converting enzyme inhibitor (ACEi… (More)
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Highly Cited
1999
Highly Cited
1999
The development of congestive heart failure (CHF) is associated with left ventricle (LV) dilation and myocardial remodeling. The… (More)
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