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Org 26576

 
National Institutes of Health

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2012
2012
Org 26576 acts by modulating ionotropic AMPA-type glutamate receptors to enhance glutamatergic neurotransmission. The aim of this… Expand
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2012
2012
BACKGROUND It has been posited that glutamate dysregulation contributes to the pathophysiology of attention-deficit/hyperactivity… Expand
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2012
2012
BackgroundA key challenge to dose selection in early central nervous system (CNS) clinical drug development is that patient… Expand
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2012
2012
Brain derived neurotrophic factor (BDNF) is a key mediator of brain plasticity. The modulation of its expression and function is… Expand
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2011
2011
IntroductionThe α-amino-3-hydroxy-5-methylisoxazole-4-propionic acid (AMPA) receptor potentiator Org 26576 represents an… Expand
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2011
2011
RationaleThe α-amino-3-hydroxy-5-methyl-4-isoxazole propionic acid (AMPA) receptor positive allosteric modulators (AMPA-PAMs… Expand
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2009
2009
Ampakines have shown beneficial effects on cognition in selected animal models of learning. However, their ability to modify long… Expand
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2009
2009
RationaleCurrently available antidepressants upregulate hippocampal neurogenesis and prefrontal gliogenesis after chronic… Expand
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2007
2007
The ionotropic glutamate AMPA ((R,S)-α-amino-3-hydroxy-5-methyl-4isoxazolepropionic acid) receptor mediates the majority of… Expand
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2005
2005
AMPA receptor potentiating drugs (e.g. ampakines) enhance glutamatergic neurotransmission, and may have potential therapeutic… Expand
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