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NS5806

National Institutes of Health

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
2017
2017
Mechanisms Underlying Epicardial Radiofrequency Ablation to Suppress Arrhythmogenesis in Experimental Models of Brugada Syndrome.
2016
2016
Neurovascular coupling protects neurons against hypoxic injury via inhibition of potassium currents by generation of nitric oxide in direct neuron and endothelium cocultures
Highly Cited
2014
Highly Cited
2014
Mechanisms underlying the development of the electrocardiographic and arrhythmic manifestations of early repolarization syndrome.
2014
2014
Modulation of the Voltage-gated Potassium Channel (Kv4.3) and the Auxiliary Protein (KChIP3) Interactions by the Current Activator NS5806*
Background: KChIP3 association with Kv4 channels regulate the K+ current gating. Results: NS5806 binds to KChIP3 and stabilizes… 
Highly Cited
2013
Highly Cited
2013
Cellular mechanisms underlying the effects of milrinone and cilostazol to suppress arrhythmogenesis associated with Brugada syndrome.
2013
2013
Effect of Wenxin Keli and quinidine to suppress arrhythmogenesis in an experimental model of Brugada syndrome.
2012
2012
Physiological consequences of transient outward K+ current activation during heart failure in the canine left ventricle.
2012
2012
Ionic and Cellular Mechanisms Underlying the Development of Acquired Brugada Syndrome in Patients Treated with Antidepressants
Antidepressant‐Induced Brugada Syndrome. 
Highly Cited
2010
Highly Cited
2010
Effect of the Ito activator NS5806 on cloned Kv4 channels depends on the accessory protein KChIP2
The compound NS5806 increases the transient outward current (Ito) in canine ventricular cardiomyocytes and slows current decay… 
Highly Cited
2008
Highly Cited
2008
A transient outward potassium current activator recapitulates the electrocardiographic manifestations of Brugada syndrome.
AIMS Transient outward potassium current (I(to)) is thought to be central to the pathogenesis of the Brugada syndrome (BrS… 
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