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MIRN582 microRNA, human

Known as: hsa-mir-582, miR-582, human, miR-582-5p, human 
 
National Institutes of Health

Topic mentions per year

Topic mentions per year

2013-2017
01220132017

Related topics

Related topics

1 relation

Broader (1)

MicroRNAs

Related mentions per year

Related mentions per year

1988-2019
19801990200020102020
MIRN582 microRNA, human
MicroRNAs

Papers overview

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2017
2017
MicroRNA-582-5p suppressed gastric cancer cell proliferation via targeting AKT3.
  • Ye Jin, L Tao, +4 authors S Jin
  • European review for medical and pharmacological…
  • 2017
OBJECTIVE To dissect the functioning mode of miR-582-5p on gastric cancer cell growth and provide therapeutic targets for gastric… (More)
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2017
2017
miR-582-5p inhibits invasion and migration of salivary adenoid cystic carcinoma cells by targeting FOXC1.
Objective Neurotropism of salivary adenoid cystic carcinoma (SACC) and pulmonary metastasis may lead to in treatment failure. miR… (More)
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2016
2016
MiR-582-5p/miR-590-5p targeted CREB1/CREB5–NF-κB signaling and caused opioid-induced immunosuppression in human monocytes
Chronic opioid abusers are more susceptible to bacterial and viral infections, but the molecular mechanism underlying opioid… (More)
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2015
2015
Upregulation of miR-582-5p inhibits cell proliferation, cell cycle progression and invasion by targeting Rab27a in human colorectal carcinoma
Colorectal carcinoma (CRC) is known as the most common cancer. MicroRNAs (miRNAs) have been proven to have important roles in… (More)
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2014
2014
Up-regulation of miR-582-5p regulates cellular proliferation of prostate cancer cells under androgen-deprived conditions.
BACKGROUND MicroRNAs are noncoding small RNA that negatively regulate target gene expression by binding to the 3'-UTR of mRNA… (More)
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2013
2013
miR-582-5p Is Upregulated in Patients with Active Tuberculosis and Inhibits Apoptosis of Monocytes by Targeting FOXO1
Macrophage apoptosis is a host innate defense mechanism against tuberculosis (TB). In this study, we found that percentage of… (More)
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