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MIR142 gene

Known as: MIR142, hsa-mir-142, miRNA142 
National Institutes of Health

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2 relations
MIRN142 microRNA, humanMicroRNAs

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
2017
2017
Abstract 3042:MIR142loss-of-function mutations promote leukemogenesis through derepression ofASH1Lresulting in increasedHOXgene expression
MIR142 mutations have been identified in acute myeloid leukemia (AML) and non-Hodgkins lymphoma. In AML, all MIR142 mutations… 
2015
2015
Demethylation of microRNA-142 induced by demethylation agents plays a suppressive role in osteosarcoma cells.
Osteosarcoma (OS), the most common malignant bone tumor, occurs mainly in adolescents and young adults, with a morbidity of ∼5… 
2014
2014
MicroRNA142-3p Promotes Tumor-Initiating and Radioresistant Properties in Malignant Pediatric Brain Tumors
Primary central nervous system (CNS) atypical teratoid/rhabdoid tumor (ATRT) is an extremely malignant pediatric brain tumor… 
2013
2013
Expression and prognostic significance of hsa-miR-142-3p in acute leukemias.
OBJECTIVES The microRNA 142 (miR-142) is expressed at high levels in mature hematopoietic cells and has a crucial role during T… 
2013
2013
Expression and prognostic significance of hsa-miR-1423 p in acute leukemias
Objectives: The microRNA 142 (miR-142) is expressed at high levels in mature hematopoietic cells and has a crucial role during T… 
2011
2011
MicroRNAs as Effectors for Acetyltransferase p300 in Cardiac Hypertrophy
of a dissertation at the University of Miami. Dissertation supervised by Professor Nanette H. Bishopric. No. of pages in text… 
2010
2010
[A novel method for monitoring miRNA activity by expression changes of secreted luciferase gene in live cells].
We developed a method for monitoring of miRNA activity in live cells by a secreted luciferase gene based plasmid sensor named as… 
2008
2008
Downregulation of microRNA-142 by proto-oncogene LMO2 and its co-factors
in the 50 UTR of BCL6, or in the distant ABR. Thus, in our cases BCL6 deregulation can not be explained by promoter substitution… 
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