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Long-Term Synaptic Depression

Known as: Long Term Depression, Neurophysiological, Neurophysiological Long-Term Depressions, Physiologic Long-Term Depression 
A persistent activity-dependent decrease in synaptic efficacy between NEURONS. It typically occurs following repeated low-frequency afferent… Expand
National Institutes of Health

Papers overview

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Review
2018
Review
2018
Changes in the properties and postsynaptic abundance of AMPA-type glutamate receptors (AMPARs) are major mechanisms underlying… Expand
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Review
2017
Review
2017
BACKGROUND In recent years, many studies have evaluated the effects of noninvasive brain stimulation (NIBS) techniques for the… Expand
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Highly Cited
1998
Highly Cited
1998
  • G. Bi, M. Poo
  • The Journal of Neuroscience
  • 1998
  • Corpus ID: 16317084
In cultures of dissociated rat hippocampal neurons, persistent potentiation and depression of glutamatergic synapses were induced… Expand
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Highly Cited
1998
Highly Cited
1998
Brief bath application of N-methyl-D-aspartate (NMDA) to hippocampal slices produces long-term synaptic depression (LTD) in CA1… Expand
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Highly Cited
1994
Highly Cited
1994
LONG-TERM potentiation (LTP) is a synaptic mechanism thought to be involved in learning and memory1. Long-term depression (LTD… Expand
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Review
1994
Review
1994
Long-term potentiation (LTP) is a synaptic enhancement that follows brief, high-frequency electrical stimulation in the… Expand
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Review
1993
Review
1993
In many brain areas, including the cerebellar cortex, neocortex, hippocampus, striatum and nucleus accumbens, brief activation of… Expand
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Highly Cited
1992
Highly Cited
1992
The mechanisms responsible for long-lasting, activity-dependent decreases in synaptic efficacy are not well understood. We have… Expand
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Highly Cited
1990
Highly Cited
1990
IN the hippocampus and neocortex, high-frequency (tetanic) stimu-lation of an afferent pathway leads to long-term potentiation… Expand
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Review
1989
Review
1989
  • M. Ito
  • Annual review of neuroscience
  • 1989
  • Corpus ID: 25281897
LTD has now been established as a synaptic plasticity specific to the cerebellum. Cellular and molecular mechanisms of LTD have… Expand
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