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Hypoaldosteronism, Hyporeninemic

Known as: Acidosis, Renal Tubular Type IV, Type IV Renal Tubular Acidosis, Renal Tubular Acidosis, Type IV 
A selective aldosterone deficiency resulting from diminished RENIN-generated ANGIOTENSIN II, a key stimulus to aldosterone secretion. Previously, it… Expand
National Institutes of Health

Papers overview

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Review
2013
Review
2013
Sodium and potassium are essential for human health. They are important ions in the body and are associated with many physiologic… Expand
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Review
2004
Review
2004
  • P. White
  • Molecular and Cellular Endocrinology
  • 2004
  • Corpus ID: 44778332
Aldosterone's main actions are to regulate intravascular volume and serum electrolytes by controlling sodium absorbtion and… Expand
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1995
1995
The effects of chronic stress on the renin-angiotensin-aldosterone system were studied by analysis of plasma hormone levels… Expand
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Review
1993
Review
1993
NSAIDs pose little threat of renal insult in normal, healthy persons at therapeutic dosages. However, NSAID administration to… Expand
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Highly Cited
1987
Highly Cited
1987
Four patients with the acquired immune deficiency syndrome (AIDS) and persistent unexplained hyperkalemia were studied. Testing… Expand
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1986
1986
Hyporeninemic hypoaldosteronism is an important cause of hyperkalemia and is characterized by low renin secretion. We found that… Expand
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Highly Cited
1981
Highly Cited
1981
To define the changes in adrenal gland function during critical illness, we evaluated 28 severely ill patients with persistent… Expand
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Highly Cited
1980
Highly Cited
1980
Presentation of the Forum is made possible by grants from Hoechst-Roussel Pharmaceuticals Inc., Smith Kline & French Laboratories… Expand
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Highly Cited
1979
Highly Cited
1979
Hyporeninemic hypoaldosteronism was diagnosed in a young woman with glomerulonephritis who was receiving indomethacin therapy… Expand
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Highly Cited
1977
Highly Cited
1977
In four patients with renal hyperchloremic acidosis and hyperkalemia, hyporeninemic hypoaldosteronism and chronic renal… Expand
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