Skip to search formSkip to main content
You are currently offline. Some features of the site may not work correctly.

Hyperphosphaturia

Known as: Phosphaturia 
An increased excretion of phosphates in the urine. [HPO:curators]
National Institutes of Health

Related topics

Related topics

6 relations
Cytochrome-c Oxidase DeficiencyDent disease 1Hepatolenticular DegenerationHyperphosphatemia (disorder)
Expand

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
Highly Cited
2011
Highly Cited
2011
Klotho deficiency causes vascular calcification in chronic kidney disease.
Soft-tissue calcification is a prominent feature in both chronic kidney disease (CKD) and experimental Klotho deficiency, but… Expand
  • figure 1
  • table 1
  • table 2
  • figure 2
  • figure 3
Highly Cited
2011
Highly Cited
2011
Vegetarian compared with meat dietary protein source and phosphorus homeostasis in chronic kidney disease.
BACKGROUND AND OBJECTIVES Patients with advanced chronic kidney disease (CKD) are in positive phosphorus balance, but phosphorus… Expand
  • figure 1
  • table 1
  • table 2
  • figure 2
  • figure 3
Highly Cited
2010
Highly Cited
2010
Klotho: a novel phosphaturic substance acting as an autocrine enzyme in the renal proximal tubule
Klotho has profound effects on phosphate metabolism, but the mechanisms of how Klotho affects phosphate homeostasis is unknown… Expand
Highly Cited
2010
Highly Cited
2010
PTH increases FGF23 gene expression and mediates the high-FGF23 levels of experimental kidney failure: a bone parathyroid feedback loop.
Parathyroid hormone (PTH) and fibroblast growth factor 23 (FGF23) target the kidney to cause a phosphaturia. FGF23 also acts on… Expand
  • figure 1
  • table 1
  • figure 2
  • figure 3
  • figure 4
Highly Cited
2006
Highly Cited
2006
DMP1 mutations in autosomal recessive hypophosphatemia implicate a bone matrix protein in the regulation of phosphate homeostasis
Hypophosphatemia is a genetically heterogeneous disease. Here, we mapped an autosomal recessive form (designated ARHP) to… Expand
  • figure 1
  • table 1
Highly Cited
2005
Highly Cited
2005
Fibroblast growth factor-23 mitigates hyperphosphatemia but accentuates calcitriol deficiency in chronic kidney disease.
Hyperphosphatemia, calcitriol deficiency, and secondary hyperparathyroidism (SHPT) are common complications of chronic kidney… Expand
  • table 1
  • table 2
  • figure 1
  • figure 2
  • figure 3
Review
1992
Review
1992
Hyperphosphatemia: its consequences and treatment in patients with chronic renal disease.
Control of phosphorus accumulation in chronic renal insufficiency is crucial to the prevention of secondary hyperparathyroidism… Expand
Highly Cited
1973
Highly Cited
1973
Effects of parathyroid hormone on renal tubular reabsorption of calcium, sodium, and phosphate.
AGUS, ZALMAN S., LAURENCE B. GARDNER, LAURENCE H. BECK, AND MARTIN GOLDBERG. Efects of parathyroid hormone on renal tubular… Expand
  • figure 1
  • figure 2
  • figure 3
  • figure 4
Highly Cited
1972
Highly Cited
1972
Renal handling of calcium and phosphate during mineralocorticoid "escape" in man.
Renal handling of calcium and phosphate during mineralocorticoid "escape" in man. Previous work in animals and man suggests a… Expand
Highly Cited
1970
Highly Cited
1970
Effects of parathyroid hormone on plasma and urinary adenosine 3',5'-monophosphate in man.
The effects of parathyroid hormone (PTH) on plasma and urinary adenosine 3',5'-monophosphate (cyclic AMP) levels were studied in… Expand
  • figure 1
  • figure 2
  • figure 3
  • table I
  • figure 4
By clicking accept or continuing to use the site, you agree to the terms outlined in our Privacy Policy, Terms of Service, and Dataset License
ACCEPT & CONTINUE