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HL 004

Known as: HL-004 
National Institutes of Health

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Acetanilides

Papers overview

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Highly Cited
2002
Highly Cited
2002
Macrophage Plasma Membrane Cholesterol Contributes to Brucella abortus Infection of Mice
ABSTRACT Brucella abortus is a facultative intracellular bacterium capable of surviving inside macrophages. Intracellular… 
1998
1998
Stimulation of cholesterol release from rabbit foam cells by the action of a new inhibitor for acyl CoA:cholesterol acyltransferase (ACAT), HL-004.
A new inhibitor of acyl CoA:cholesterol acyltransferase (ACAT), HL-004 [N-(2, 6-diisopropylphenyl)tetradecylthioacetamide… 
1998
1998
HL-004, the ACAT inhibitor, prevents the progression of atherosclerosis in cholesterol-fed rabbits.
1998
1998
The ACAT inhibitor HL-004 inhibits cholesterol absorption and lowers serum cholesterol in rats.
1997
1997
ACAT inhibitor HL-004 accelerates the regression of hypercholesterolemia in stroke-prone spontaneously hypertensive rats (SHRSP): stimulation of bile acid production by HL-004.
1996
1996
Effect of the ACAT inhibitor, HL-004, on cholesterol metabolism in macrophages.
1996
1996
The hypolipidemic action of the ACAT inhibitor HL-004 in hamsters fed normal chow.
1995
1995
Hypolipidemic and antiatherosclerotic effects of a novel ACAT inhibitor, HL-004, in stroke-prone spontaneously hypertensive rats.
The hypolipidemic and antiatherosclerotic effects of a novel acyl CoA:cholesterol acyltransferase (ACAT) inhibitor, HL-004, were… 
1995
1995
Effects of HL-004, a novel ACAT inhibitor, on cholesterol accumulation and removal in cultured smooth muscle cells from stroke-prone spontaneously hypertensive rats (SHRSP).
1995
1995
Peritoneal macrophages from stroke-prone spontaneously hypertensive rats accumulate more cholesteryl ester than do macrophages from Wistar-Kyoto rats.
The intracellular cholesterol metabolism of peritoneal macrophages from stroke-prone spontaneously hypertensive rats (SHRSP) was… 
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