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GOLGA6L2 gene

Known as: CT105, GOLGA6L2, golgin A6 family-like 2 
National Institutes of Health

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
2019
2019
Abstract CT105: AUTO1, a novel fast off CD19CAR delivers durable remissions and prolonged CAR T cell persistence with low CRS or neurotoxicity in adult ALL
Introduction: CD19 CAR T therapy has advanced treatment of relapsed/refractory (r/r) Acute Lymphoblastic Leukemia (B-ALL… 
2013
2013
Angelman syndrome and severe infections in a patient with de novo 15q11.2-q13.1 deletion and maternally inherited 2q21.3 microdeletion.
2009
2009
Selective apoptosis induction in the hippocampal mossy fiber pathway by exposure to CT105, the C-terminal fragment of Alzheimer's amyloid precursor protein
2004
2004
The Effects of KongJin-Dan(KJD) on the Alzheimer's Disease Model Induced by CT105
Objective : This experiment was designed to investigate the effect of KongJin-dan(KJD) on the Alzheimer's disease. Method : The… 
2002
2002
Molecular mechanisms underlying cyclic AMP inhibition of macrophage dependent TNF-α production and neurotoxicity in response to amyloidogenic C-terminal fragment of Alzheimer's amyloid precursor…
2002
2002
Molecular mechanisms underlying cyclic AMP inhibition of macrophage dependent TNF-alpha production and neurotoxicity in response to amyloidogenic C-terminal fragment of Alzheimer's amyloid precursor…
In the present study, we characterized the intracellular pathway involved in the macrophage production of tumor necrosis factor… 
2002
2002
The Effects of Crataegus prinnatiflora BGE. var. major N.E. BR(CPVM) Extract on the the Alzheimer's Disease Model Induced by CT105
This research investigates the effect of the Crataegus pinnatifida BGE. var. major N.E. BR(CPVM) on Alzheimer's disease… 
Highly Cited
2001
Highly Cited
2001
Memory impairment and cholinergic dysfunction by centrally administered Aβ and carboxyl‐terminal fragment of Alzheimer's APP in mice
Alzheimer's disease (AD) is characterized by cognitive impairment and excessive deposition of neurite plaques (NPs). Although the… 
2001
2001
Effects of the carboxyl‐terminal fragment of Alzheimer's amyloid precursor protein and amyloid β ‐peptide on the production of cytokines and nitric oxide in glial cells
In a brain with Alzheimer's disease (AD), activated glial cells are observed surrounding amyloid plaque. In this study, to… 
2000
2000
Roles of A beta and carboxyl terminal peptide fragments of amyloid precursor protein in Alzheimer disease.
Several lines of evidence indicate that A beta may play an important role in the pathogenesis of AD. However, there are several… 
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