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GKT 136901

Known as: GKT-136901, GKT136901 
 
National Institutes of Health

Papers overview

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2019
2019
Background NADPH oxidases (NOX) are a family of flavoenzymes that catalyze the formation of superoxide anion radical (O2•-) and… Expand
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2019
2019
Dysfunctional reactive oxygen species (ROS) signaling is considered an important disease mechanism. Therapeutically, non… Expand
Review
2017
Review
2017
The NADPH oxidase (NOX) family of enzymes produces ROS as their sole function and is becoming recognized as key modulators of… Expand
2017
2017
HighlightsPharmacological NOX4 inhibition is inefficient in later stages of neuropathic pain.Nox4 is upregulated early after… Expand
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2015
2015
OBJECTIVES Osteoarthritis (OA) is characterized by a progressive alteration of the biochemical properties of the articular… Expand
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2015
2015
Abstract NADPH oxidases (NOXs) represent the only known dedicated source of reactive oxygen species (ROS) and thus a prime… Expand
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2014
2014
NADPH oxidases (NOX), catalyzing the reduction of molecular oxygen to form the superoxide radical anion (•O₂⁻) and hydrogen… Expand
Highly Cited
2013
Highly Cited
2013
Nox (NADPH oxidase)-derived ROS (reactive oxygen species) have been implicated in the development of diabetic nephropathy. Of the… Expand
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Highly Cited
2010
Highly Cited
2010
The intracellular signaling events by which NADPH oxidase-generated reactive oxygen species (ROS) modulate vascular smooth muscle… Expand
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Highly Cited
2006
Highly Cited
2006
P300-based GKT (guilty knowledge test) has been suggested as an alternative approach for conventional polygraphy. The purpose of… Expand
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