Skip to search formSkip to main content
You are currently offline. Some features of the site may not work correctly.

FAM64A gene

Known as: RCS1, FLJ10491, FLJ10156 
 
National Institutes of Health

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
2019
2019
FAM64A, a marker of cell proliferation, has been investigated as a potential biomarker in several cancers. In the present study… Expand
  • table 1
  • table 1
  • figure 1
  • figure 2
  • table 2
2019
2019
Clear cell renal cell carcinoma (ccRCC) is the most common and lethal renal malignant tumor in adults. The aim of the present… Expand
  • table I
  • figure 1
  • figure 2
  • figure 3
  • figure 4
2019
2019
Significance The transcription factor STAT3 plays pivotal roles in various physiological processes, including differentiation of… Expand
  • figure 1
  • figure 2
  • figure 3
  • figure 4
  • figure 5
2018
2018
In the human genome, miR‐451a, miR‐144‐5p (passenger strand), and miR‐144‐3p (guide strand) reside in clustered microRNA (miRNA… Expand
2017
2017
Fetal cardiomyocytes actively proliferate to form the primitive heart in utero in mammals, but they stop dividing shortly after… Expand
  • figure 1
  • table 1
  • figure 2
  • figure 3
  • table 2
2017
2017
Both tumor and adjacent normal tissues are valuable in cancer research. Transcriptional response profiles represent the changes… Expand
2016
2016
The CATS (FAM64A) protein interacts with CALM (PICALM) and the leukemic fusion protein CALM/AF10. CATS is highly expressed in… Expand
  • figure 1
  • figure 2
  • figure 3
  • figure 4
  • figure 5
2013
2013
The CATS protein (also known as FAM64A and RCS1) was first identified as a novel CALM (PICALM) interactor that influences the… Expand
  • figure 2
  • figure 3
  • figure 4
  • figure 5
Highly Cited
2009
Highly Cited
2009
Aims: To obtain an insight into the function of cellular prion protein (PrPC), we studied PrPC‐interacting proteins (PrPIPs) by… Expand
2008
2008
The anaphase-promoting complex/cyclosome (APC/C) controls the onset of anaphase by targeting securin for destruction. We report… Expand
  • figure 1
  • figure 2
  • figure 3
  • figure 4
  • figure 5