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CGP 3466B

Known as: CGP-3466B, CGP3466B 
National Institutes of Health

Papers overview

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2018
2018
Blocking the nitrosylation of GAPDH may reduce amyloid-induced tau toxicity in the brain and slow the progression of Alzheimer’s… 
2017
2017
Neuronal apoptosis chiefly contributes to the cell loss following traumatic brain injury (TBI). CGP3466B is a compound related to… 
Highly Cited
2016
Highly Cited
2016
Significance Cocaine is one of the most abused drugs in modern society, with overdoses that are frequently lethal. Molecular… 
2006
2006
Aims/hypothesisPost-translational modifications, such as isomerisation of native proteins, may create new antigenic epitopes and… 
2004
2004
BACKGROUND: There is an accumulating body of evidence that apoptosis is involved in the motor neuron death that occurs in ALS… 
Highly Cited
2000
Highly Cited
2000
The ability of CGP 3466B to attenuate the behavioural and morphological consequences of experimentally induced cell death was… 
Highly Cited
2000
Highly Cited
2000
Apoptosis and mitochondrial dysfunction are thought to be involved in the aetiology of neurodegenerative diseases. We have tested… 
2000
2000
Abstract. The propargylamine derivative CGP 3466 (dibenzo[b,f]oxepin-10-ylmethyl-methyl-prop-2-ynyl-amine) has previously been… 
2000
2000
The propargylamine CGP 3466B prevents dopamine cell death both in vitro and in rodent models of Parkinson's disease. The present…