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CGP 3466B

Known as: CGP-3466B, CGP3466B 
 
National Institutes of Health

Papers overview

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2018
2018
Blocking the nitrosylation of GAPDH may reduce amyloid-induced tau toxicity in the brain and slow the progression of Alzheimer’s… Expand
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2017
2017
Neuronal apoptosis chiefly contributes to the cell loss following traumatic brain injury (TBI). CGP3466B is a compound related to… Expand
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2016
2016
Significance Cocaine is one of the most abused drugs in modern society, with overdoses that are frequently lethal. Molecular… Expand
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2013
2013
BACKGROUND Mammalian sterile 20-like kinase 1 (Mst1) is a mammalian homolog of Hippo kinase from Drosophila and it is a critical… Expand
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2006
2006
Aims/hypothesisPost-translational modifications, such as isomerisation of native proteins, may create new antigenic epitopes and… Expand
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2004
2004
BACKGROUND: There is an accumulating body of evidence that apoptosis is involved in the motor neuron death that occurs in ALS… Expand
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Highly Cited
2000
Highly Cited
2000
The ability of CGP 3466B to attenuate the behavioural and morphological consequences of experimentally induced cell death was… Expand
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Highly Cited
2000
Highly Cited
2000
Apoptosis and mitochondrial dysfunction are thought to be involved in the aetiology of neurodegenerative diseases. We have tested… Expand
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2000
2000
Abstract. The propargylamine derivative CGP 3466 (dibenzo[b,f]oxepin-10-ylmethyl-methyl-prop-2-ynyl-amine) has previously been… Expand
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2000
2000
The propargylamine CGP 3466B prevents dopamine cell death both in vitro and in rodent models of Parkinson's disease. The present… Expand
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