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CGP 3466B

Known as: CGP-3466B, CGP3466B 
National Institutes of Health

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
2018
2018
Blocking the nitrosylation of GAPDH may reduce amyloid-induced tau toxicity in the brain and slow the progression of Alzheimer’s… Expand
2016
2016
Significance Cocaine is one of the most abused drugs in modern society, with overdoses that are frequently lethal. Molecular… Expand
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2013
2013
BACKGROUND Mammalian sterile 20-like kinase 1 (Mst1) is a mammalian homolog of Hippo kinase from Drosophila and it is a critical… Expand
2013
2013
Cocaine's behavioral-stimulant effects derive from potentiation of synaptic signaling by dopamine and serotonin leading to… Expand
2006
2006
Aims/hypothesisPost-translational modifications, such as isomerisation of native proteins, may create new antigenic epitopes and… Expand
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2004
2004
BACKGROUND: There is an accumulating body of evidence that apoptosis is involved in the motor neuron death that occurs in ALS… Expand
Highly Cited
2000
Highly Cited
2000
The ability of CGP 3466B to attenuate the behavioural and morphological consequences of experimentally induced cell death was… Expand
2000
2000
The propargylamine CGP 3466B prevents dopamine cell death both in vitro and in rodent models of Parkinson's disease. The present… Expand
2000
2000
Abstract. The propargylamine derivative CGP 3466 (dibenzo[b,f]oxepin-10-ylmethyl-methyl-prop-2-ynyl-amine) has previously been… Expand
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Highly Cited
2000
Highly Cited
2000
Apoptosis and mitochondrial dysfunction are thought to be involved in the aetiology of neurodegenerative diseases. We have tested… Expand