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Atosiban

Known as: 1-deamino-2-Tyr(OEt)-4-Thr-8-Orn-oxytocin, (Mpa(1)-D-Tyr(Et)(2)-Thr(4)-Orn(8))-oxytocin, 1-Deamino-2D-tyr-(OEt)-4-thr-8-orn-oxytocin 
National Institutes of Health

Related topics

Related topics

6 relations
Atosiban Acetateanalogs & derivatives

Broader (3)

Hormone AntagonistsTocolytic AgentsVasotocin

Narrower (1)

RWJ-22164

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
Highly Cited
2014
Highly Cited
2014
Oxytocin via its receptor affects restraint stress-induced methamphetamine CPP reinstatement in mice: Involvement of the medial prefrontal cortex and dorsal hippocampus glutamatergic system
Review
2014
Review
2014
Oxytocin receptor antagonists for inhibiting preterm labour.
BACKGROUND Preterm birth, defined as birth before 37 completed weeks, is the single most important cause of perinatal mortality… 
2007
2007
The present and future of tocolysis
2007
2007
Oxytocin--a stimulator of directed sperm transport in humans.
2007
2007
Atosiban versus usual care for the management of preterm labor
Abstract Objective: To compare the efficacy of atosiban with usual management of threatened preterm labor. Methods: In this… 
2006
2006
Role of intraluteal prostaglandin F(2alpha), progesterone and oxytocin in basal and pulsatile progesterone release from developing bovine corpus luteum.
2004
2004
Centrally administered oxytocin elicits exaggerated grooming in oxytocin null mice
2004
2004
A prospective randomised trial of atosiban versus hexoprenaline for acute tocolysis and intrauterine resuscitation
Objective  The aim of this study was to compare the efficacy and side effect profile of atosiban with hexoprenaline when used for… 
Review
2003
Review
2003
Effectiveness and safety of the oxytocin antagonist atosiban versus beta‐adrenergic agonists in the treatment of preterm labour
1546 women) from a population of nearly 1.4 million. It is the biggest published cross sectional survey on this topic that we can… 
1999
1999
Oxytocin and vasopressin constrict rat isolated uterine resistance arteries by activating vasopressin V1A receptors.
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