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AS 601245

Known as: AS-601245, AS601245 
 
National Institutes of Health

Papers overview

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Review
2018
Review
2018
In this article, we review the literature regarding the role of c-Jun N-terminal kinases (JNKs) in cerebral and myocardial… Expand
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Highly Cited
2012
Highly Cited
2012
BackgroundWhite matter injury is the major form of brain damage in very preterm infants. Selective white matter injury in the… Expand
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Highly Cited
2010
Highly Cited
2010
BackgroundApoptosis, neuroinflammation and blood-brain barrier (BBB) damage affect the susceptibility of the developing brain to… Expand
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2009
2009
Hepatocellular carcinoma (HCC) displays a striking resistance to tumor necrosis factor-related apoptosis-inducing ligand (TRAIL… Expand
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2008
2008
Based on their proven ability, in animal models of stroke, to reduce damage to brain grey matter, many drugs have been tested in… Expand
Highly Cited
2007
Highly Cited
2007
Defects in the proton-translocating NADH-quinone oxidoreductase (complex I) of mammalian mitochondria are linked to… Expand
Highly Cited
2005
Highly Cited
2005
Several lines of evidence support the hypothesis that c-Jun N-terminal kinase (JNKs) plays a critical role in a wide range of… Expand
Highly Cited
2005
Highly Cited
2005
c‐Jun N‐terminal kinase (JNK), a member of the mitogen‐activated protein kinase family, is activated in response to a number of… Expand
Highly Cited
2004
Highly Cited
2004
Recent evidence suggests that activation of the c-Jun NH2-terminal protein kinase (JNK) signal transduction pathway may play a… Expand
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Highly Cited
2004
Highly Cited
2004
Myocardial ischemia/reperfusion is associated with inflammation, apoptosis and necrosis. During this process, c‐jun N‐terminal… Expand