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AS 601245

Known as: AS-601245, AS601245 
National Institutes of Health

Papers overview

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Review
2020
Review
2020
Abstract c-Jun N-terminal kinase (JNK) signalling regulates both cancer cell apoptosis and survival. Emerging evidence show that… 
2014
2014
BackgroundHypoxic-ischemia (HI) and inflammation are the two major pathogenic mechanisms of brain injury in very preterm infants… 
Highly Cited
2012
Highly Cited
2012
BackgroundWhite matter injury is the major form of brain damage in very preterm infants. Selective white matter injury in the… 
Highly Cited
2010
Highly Cited
2010
BackgroundApoptosis, neuroinflammation and blood-brain barrier (BBB) damage affect the susceptibility of the developing brain to… 
Highly Cited
2005
Highly Cited
2005
Several lines of evidence support the hypothesis that c-Jun N-terminal kinase (JNKs) plays a critical role in a wide range of… 
Highly Cited
2005
Highly Cited
2005
c‐Jun N‐terminal kinase (JNK), a member of the mitogen‐activated protein kinase family, is activated in response to a number of… 
Highly Cited
2004
Highly Cited
2004
Recent evidence suggests that activation of the c-Jun NH2-terminal protein kinase (JNK) signal transduction pathway may play a… 
Highly Cited
2004
Highly Cited
2004
Myocardial ischemia/reperfusion is associated with inflammation, apoptosis and necrosis. During this process, c‐jun N‐terminal…