AS 601245

Known as: AS-601245, AS601245 
 
National Institutes of Health

Topic mentions per year

Topic mentions per year

2004-2017
012320042017

Papers overview

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2015
2015
c-Jun N-terminal kinase (JNK) plays a key role in the regulation of neuronal apoptosis. Previous studies have revealed that… (More)
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2014
2014
Hypoxic-ischemia (HI) and inflammation are the two major pathogenic mechanisms of brain injury in very preterm infants. The… (More)
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2013
2013
The human kinome comprises over 800 individual kinases. These contribute in multiple ways to regulation of cellular metabolism… (More)
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2012
2012
Despite its clinical importance, the molecular biology of HIV-1 latency control is at best partially understood, and the… (More)
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2011
2011
c-Jun N-terminal kinase (JNK) pathway has been shown to be essential for cell cycle progression and mitosis. We previously showed… (More)
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2010
2010
Apoptosis, neuroinflammation and blood-brain barrier (BBB) damage affect the susceptibility of the developing brain to hypoxic… (More)
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2009
2009
Hepatocellular carcinoma (HCC) displays a striking resistance to tumor necrosis factor-related apoptosis-inducing ligand (TRAIL… (More)
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2008
2008
BACKGROUND AND PURPOSE Based on their proven ability, in animal models of stroke, to reduce damage to brain grey matter, many… (More)
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Highly Cited
2004
Highly Cited
2004
1 Myocardial ischemia/reperfusion is associated with inflammation, apoptosis and necrosis. During this process, c-jun N-terminal… (More)
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2004
2004
Recent evidence suggests that activation of the c-Jun NH2-terminal protein kinase (JNK) signal transduction pathway may play a… (More)
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