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API5 gene

Known as: API5L1, AAC11, AAC-11 
National Institutes of Health

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1 relation
API5 protein, human

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
2016
2016
A Cell-Penetrating Peptide Targeting AAC-11 Specifically Induces Cancer Cells Death.
AAC-11 is an antiapoptotic protein that is upregulated in most cancer cells. Increased expression of AAC-11 confers a survival… 
2015
2015
Nucleoprotein of influenza A virus negatively impacts antiapoptotic protein API5 to enhance E2F1-dependent apoptosis and virus replication
Apoptosis of host cells profoundly influences virus propagation and dissemination, events that are integral to influenza A virus… 
2015
2015
Common variants in or near ZNRF1, COLEC12, SCYL1BP1 and API5 are associated with diabetic retinopathy in Chinese patients with type 2 diabetes
Aims/hypothesisThree recent genome-wide association studies (GWAS) identified several single-nucleotide polymorphisms (SNPs) with… 
2015
2015
Tumor suppressors miR-143 and miR-145 and predicted target proteins API5, ERK5, K-RAS, and IRS-1 are differentially expressed in proximal and distal colon.
The colon differs regionally in local luminal environment, excretory function, and gene expression. Polycistronic microRNA (miR… 
2014
2014
MiR-224 expression increases radiation sensitivity of glioblastoma cells.
2013
2013
Api5 Contributes to E2F1 Control of the G1/S Cell Cycle Phase Transition
Background The E2f transcription factor family has a pivotal role in controlling the cell fate in general, and in particular… 
Highly Cited
2009
Highly Cited
2009
The antiapoptotic protein AAC‐11 interacts with and regulates Acinus‐mediated DNA fragmentation
The nuclear factor Acinus has been suggested to mediate apoptotic chromatin condensation after caspase cleavage. However, this… 
2007
2007
The antiapoptotic protein Api5 and its partner, high molecular weight FGF2, are up‐regulated in B cell chronic lymphoid leukemia
The antiapoptotic protein Api5 and its partner, high molecular weight FGF2, were described to be up-regulated in B cell chronic… 
Highly Cited
2000
Highly Cited
2000
AAC-11 Overexpression Induces Invasion and Protects Cervical Cancer Cells from Apoptosis
To identify the genes involved in cervical carcinogenesis, we applied the mRNA differential display (DD) method to analyze normal… 
Highly Cited
1997
Highly Cited
1997
AAC-11, a novel cDNA that inhibits apoptosis after growth factor withdrawal.
Many growth factors and cytokines act as cellular survival factors by preventing programmed cell death (apoptosis). However, the… 
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