Skip to search formSkip to main content
You are currently offline. Some features of the site may not work correctly.

ANKRD55 gene

Known as: ANKYRIN REPEAT DOMAIN-CONTAINING PROTEIN 55, ankyrin repeat domain 55, FLJ11795 
 
National Institutes of Health

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
2018
2018
Recent genetic studies have identified alleles associated with opposite effects on adiposity and risk of type 2 diabetes. We… Expand
  • table 1
  • table 2
2017
2017
Single nucleotide polymorphisms (SNPs) in TNFSF4 and ANKRD55 genes have been shown to be associated with several autoimmune… Expand
  • table 1
  • table 2
Highly Cited
2016
Highly Cited
2016
Genome-wide association studies (GWAS) have identified thousands of genetic variants associated with human complex traits… Expand
  • table 4
  • figure 2
  • table 1
  • table 6
  • figure 4
Highly Cited
2016
Highly Cited
2016
IMPORTANCE Posttraumatic stress disorder (PTSD) is a prevalent, serious public health concern, particularly in the military. The… Expand
  • table 1
  • table 2
  • figure 1
  • figure 2
  • table 3
2016
2016
Genetic polymorphisms within the HLA region explain only a modest proportion of anti–cyclic citrullinated peptide (anti‐CCP… Expand
2016
2016
An intronic variant in ANKRD55, rs6859219, is a genetic risk factor for multiple sclerosis, but the biological reasons underlying… Expand
  • figure 1
  • table I
  • table II
  • table III
  • figure 2
2013
2013
CONTEXT Recently, 10 novel type 2 diabetes (T2D) susceptibility single nucleotide polymorphisms (SNPs) in ZMIZ1, ANK1, KLHDC5… Expand
2013
2013
Multiple sclerosis (MS) is a genetically complex disease that shares a substantial proportion of risk loci with other autoimmune… Expand
Highly Cited
2012
Highly Cited
2012
To extend understanding of the genetic architecture and molecular basis of type 2 diabetes (T2D), we conducted a meta-analysis of… Expand
  • figure 1
  • figure 2
  • figure 3
2012
2012
Multiple sclerosis (MS) shares some risk genes with other disorders hallmarked by an autoimmune pathogenesis, most notably IL2RA… Expand
  • table 1
  • table 2
  • figure 1