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tertiapin

 
National Institutes of Health

Papers overview

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2012
2012
Highlights ► miR-191 expression is upregulated in senescencent human epidermal keratinocytes. ► miR-191 overexpression is… Expand
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Highly Cited
2012
Highly Cited
2012
Rationale: Activation of the mitochondrial ATP-sensitive potassium channel (mitoKATP) has been implicated in the mechanism of… Expand
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Highly Cited
2006
Highly Cited
2006
It is well known that vagal nerve tone plays a crucial role in atrial fibrillation (AF). Acetylcholine-activated potassium… Expand
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Highly Cited
2005
Highly Cited
2005
Background— The molecular mechanism of increased background inward rectifier current (IK1) in atrial fibrillation (AF) is not… Expand
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Highly Cited
2005
Highly Cited
2005
Tertiapin, a short peptide from honey bee venom, has been reported to specifically block the inwardly rectifying K+ (Kir… Expand
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Highly Cited
2003
Highly Cited
2003
Endothelial cells in most vascular beds release a factor that hyperpolarizes the underlying smooth muscle, produces… Expand
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Highly Cited
2000
Highly Cited
2000
Acetylcholine (ACh) is an important neuromodulator of cardiac function that is released upon stimulation of the vagus nerve… Expand
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Highly Cited
1999
Highly Cited
1999
Tertiapin (TPN), a small protein derived from honey bee venom, inhibits the GIRK1/4 and ROMK1 channels with nanomolar affinities… Expand
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Highly Cited
1999
Highly Cited
1999
Tertiapin-Q (TPN(Q)) is a derivative of honey bee toxin tertiapin (TPN) whose methionine residue is replaced with a glutamine… Expand
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Highly Cited
1998
Highly Cited
1998
Inward-rectifier K+ channels are a group of highly specialized K+ channels that accomplish a variety of important biological… Expand
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