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proapoptotic caspase adaptor protein, human

Known as: MGC29506 protein, human, PACAP protein, human (proapoptotic) 
 
National Institutes of Health

Related topics

Related topics

1 relation

Broader (1)

Adaptor Proteins, Signal Transducing

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
2019
2019
C/EBPβ Suppresses Keratinocyte Autonomous Type 1 IFN Response and p53 to Increase Cell Survival and Susceptibility to UVB-induced Skin Cancer.
p53 is activated by DNA damage and oncogenic stimuli to regulate senescence, apoptosis and cell cycle arrest which are essential… Expand
2012
2012
Locally Different Endothelial Nitric Oxide Synthase Protein Levels in Ascending Aortic Aneurysms of Bicuspid and Tricuspid Aortic Valve
Aims. Dysregulated expression of the endothelial nitric oxide synthase (eNOS) is observed in aortic aneurysms associated with… Expand
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Highly Cited
2009
Highly Cited
2009
Transforming Growth Factor β1 Signaling via Interaction with Cell Surface Hyal-2 and Recruitment of WWOX/WOX1*
Transforming growth factor β (TGF-β) initiates multiple signal pathways and activates many downstream kinases. Here, we… Expand
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2007
2007
Generation of survival signal by differential interaction of p38MAPKalpha and p38MAPKbeta with caveolin-1 and caveolin-3 in the adapted heart.
Sphingomyelin breakdown product ceramide has recently been found to induce an adaptive response and reduce myocardial ischemia… Expand
Highly Cited
2004
Highly Cited
2004
Conformation of Membrane-associated Proapoptotic tBid*
The proapoptotic Bcl-2 family protein Bid is cleaved by caspase-8 to release the C-terminal fragment tBid, which translocates to… Expand
2004
2004
Antagonist analogue of 6-[3'-(1-adamantyl)-4'-hydroxyphenyl]-2-naphthalenecarboxylic acid (AHPN) family of apoptosis inducers that effectively blocks AHPN-induced apoptosis but not cell-cycle arrest.
The retinoid 6-[3'-(1-adamantyl)-4'-hydroxyphenyl]-2-naphthalenecarboxylic acid (AHPN) and its active analogues induce cell-cycle… Expand
Highly Cited
2003
Highly Cited
2003
The role of neurotrophic factors, apoptosis-related proteins, and endogenous antioxidants in the differential temporal vulnerability of neonatal cerebellum to ethanol.
BACKGROUND Ethanol produces abnormalities in the developing nervous system, with certain regions being vulnerable during well… Expand
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