phosphatidylinositol metabolic process

Known as: phosphatidylinositol metabolism, PtdIns metabolic process, PtdIns metabolism 
The chemical reactions and pathways involving phosphatidylinositol, any glycophospholipid in which a sn-glycerol 3-phosphate residue is esterified to… (More)
National Institutes of Health

Papers overview

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Highly Cited
2012
Highly Cited
2012
Optogenetic control of phosphoinositide metabolism.
Phosphoinositides (PIs) are lipid components of cell membranes that regulate a wide variety of cellular functions. Here we… (More)
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Highly Cited
2006
Highly Cited
2006
Two synaptojanin 1 isoforms are recruited to clathrin-coated pits at different stages.
Phosphoinositides are thought to play an important role in clathrin-coated pit (CCP) dynamics. Biochemical and structural studies… (More)
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Review
2004
Review
2004
Cell signalling through thromboxane A2 receptors.
Thromboxane A2 receptors (TPs) are widely distributed among different organ systems and have been localized on both cell… (More)
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Review
2001
Review
2001
Phosphoinositides in membrane traffic at the synapse.
Inositol phospholipids represent a minor fraction of membrane phospholipids; yet they play important regulatory functions in… (More)
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Highly Cited
1998
Highly Cited
1998
TOR2 is part of two related signaling pathways coordinating cell growth in Saccharomyces cerevisiae.
The Saccharomyces cerevisiae genes TOR1 and TOR2 encode phosphatidylinositol kinase homologs. TOR2 has two essential functions… (More)
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Highly Cited
1998
Highly Cited
1998
Activation of phospholipase C gamma by PI 3-kinase-induced PH domain-mediated membrane targeting.
Signaling via growth factor receptors frequently results in the concomitant activation of phospholipase C gamma (PLC gamma) and… (More)
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Highly Cited
1996
Highly Cited
1996
A presynaptic inositol-5-phosphatase
SYNAPTOJANIN is a nerve terminal protein of relative molecular mass 145,000 which appears to participate with dynamin in synaptic… (More)
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Highly Cited
1993
Highly Cited
1993
Mechanisms by which glucose can control insulin release independently from its action on adenosine triphosphate-sensitive K+ channels in mouse B cells.
Glucose stimulation of insulin release involves closure of ATP-sensitive K+ channels (K(+)-ATP channels), depolarization, and Ca2… (More)
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Review
1990
Review
1990
Glucose toxicity.
Glucose toxicity is a well-established entity that has been shown in animal models of diabetes to contribute to development of… (More)
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Highly Cited
1982
Highly Cited
1982
Lithium amplifies agonist-dependent phosphatidylinositol responses in brain and salivary glands.
1. The effect of Li+ on the agonist-dependent metabolism of [3H]inositol has been studied in rat brain, rat parotid and the… (More)
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