negative regulation of transforming growth factor beta1 activation

Known as: negative regulation of TGFbeta 1 activation, down-regulation of TGFbeta 1 activation, downregulation of transforming growth factor beta1 activation 
Any process that stops, prevents or reduces the frequency, rate or extent of transforming growth factor beta1 activation. [GOC:sl, GOC:TermGenie]
National Institutes of Health

Papers overview

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Highly Cited
2007
Highly Cited
2007
TGF-beta1 is a regulatory cytokine with a pleiotropic role in immune responses. TGF-beta1 is widely expressed in leukocytes and… (More)
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Highly Cited
2005
Highly Cited
2005
Human cardiac fibroblasts are the main source of cardiac fibrosis associated with cardiac hypertrophy and heart failure… (More)
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Highly Cited
2003
Highly Cited
2003
Myofibroblast differentiation and activation by transforming growth factor-beta1 (TGF-beta1) is a critical event in the… (More)
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Highly Cited
2003
Highly Cited
2003
Tubulointerstitial fibrosis is the final common result of a variety of progressive injuries leading to chronic renal failure… (More)
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Highly Cited
2002
Highly Cited
2002
Ingestion of apoptotic cells in vitro by macrophages induces TGF-beta1 secretion, resulting in an anti-inflammatory effect and… (More)
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Highly Cited
2002
Highly Cited
2002
CD4(+)CD25(+) regulatory T cells inhibit organ-specific autoimmune diseases induced by CD4(+)CD25(-) T cells and are potent… (More)
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Highly Cited
2000
Highly Cited
2000
The activin receptor-like kinase 1 (ALK1) is a type I receptor for transforming growth factor-beta (TGF-beta) family proteins… (More)
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Highly Cited
2000
Highly Cited
2000
The myofibroblast is responsible for the generation of contractile force associated with wound contraction and pathological… (More)
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Highly Cited
2000
Highly Cited
2000
When C2C12 pluripotent mesenchymal precursor cells are treated with transforming growth factor beta1 (TGF-beta1), terminal… (More)
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Highly Cited
1997
Highly Cited
1997
Transforming growth factor (TGF)-beta1 has been implicated in the pathogenesis of fibrosis based upon its matrix-inducing effects… (More)
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