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National Institutes of Health
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Excessive interferon-α signaling in autoimmunity alters glycosphingolipid processing in B cells.
Journal of autoimmunity
Corpus ID: 4865084
Excessive interferon-α (IFN-α) production by innate immune cells is a hallmark of autoimmune diseases. What other cell type…
Depletion of globosides and isoglobosides fully reverts the morphologic phenotype of Fabry disease.
Lorenz L. Lehmann
Cell and Tissue Research
Corpus ID: 14741344
Fabry disease is a monogenic X-linked lysosomal storage disease caused by α-galactosidase A (αGalA) deficiency. Enzyme…
Globosides but Not Isoglobosides Can Impact the Development of Invariant NKT Cells and Their Interaction with Dendritic Cells
The Journal of Immunology
Corpus ID: 13919276
Recognition of endogenous lipid Ag(s) on CD1d is required for the development of invariant NKT (iNKT) cells…
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