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galloflavin

 
National Institutes of Health

Papers overview

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Review
2017
Review
2017
Cancer cells reprogram their metabolism to meet the requirement for survival and rapid growth. One hallmark of cancer metabolism… Expand
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2016
2016
Activation of hepatic stellate cells (HSCs) is characterized by expression of extracellular matrix and loss of adipogenic… Expand
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2015
2015
High rates of aerobic glycolysis represent a key mechanism by which endometrial cancer cells consume glucose as its primary… Expand
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2015
2015
High-altitude residents have lower mortality rates for ischaemic heart disease and this is ascribed to cardiac gene remodelling… Expand
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2015
2015
Two flavonoids, jaceosidin 7-O-β-d-glucopyranosyl-(1→2)-β-d-glucopyranoside (1) and hispidulin 7-O-β-d-glucopyranosyl-(1→2)-β-d… Expand
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2015
2015
Altered metabolism is a critical part of cancer cell properties, but real-time monitoring of metabolomic profiles has been… Expand
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2013
2013
Lactate dehydrogenase A (LDH-A) binds single stranded DNA (ssDNA) and stimulates cell transcription. Binding is prevented by NADH… Expand
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2013
2013
Activation of the myc oncogene in cancer cells upregulates lactate dehydrogenase A (LDH-A) expression, leading to a sustained… Expand
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2012
2012
One of the most prominent alterations in cancer cells is their strict dependence on the glycolytic pathway for ATP generation… Expand
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2012
2012
Galloflavin (GF), a recently identified lactate dehydrogenase inhibitor, hinders the proliferation of cancer cells by blocking… Expand
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