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Aims Despite the lower patency of venous compared with arterial coronary artery bypass grafts, ∼50% of grafts used are saphenous… Expand Vascular smooth muscle cell (VSMC) proliferation and migration is responsible for intimal thickening that occurs in restenosis… Expand Abstract— Proteases of the plasminogen activator (PA) and matrix metalloproteinase (MMP) system play an important role in smooth… Expand Activin A alters the characteristics of human arterial smooth muscle cells (SMCs) toward a contractile, quiescent phenotype. We… Expand Background —Smooth muscle cell migration, in addition to proliferation, contributes to a large extent to the neointima formed in… Expand Gene therapy using recombinant adenoviral vectors represents a promising therapeutic tool to prevent vein graft stenosis, the… Expand Metalloproteinases (MMPs) are implicated in neointima formation and hence vein graft failure. Gene transfer to elevate local… Expand OBJECTIVES
Although cell culture techniques and animal models of intimal hyperplasia have increased our current understanding of… Expand The role of platelet-derived growth factor (PDGF), a potent vascular smooth muscle cells (SMC) mitogen and chemoattractant, was… Expand Intimal thickening is an important cause of late coronary vein graft occlusion, which no variation of surgical technique or… Expand