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U 51605

Known as: U-51605 
National Institutes of Health

Related topics

Related topics

1 relation

Broader (1)

azo analog I

Papers overview

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2015
2015
Involvement of prostaglandin I(2) in nitric oxide-induced vasodilation of retinal arterioles in rats.
2013
2013
Estradiol-17&bgr; and its Cytochrome P450- and Catechol-O-Methyltransferase–Derived Metabolites Selectively Stimulate Production of Prostacyclin in Uterine Artery Endothelial Cells: Role of Estrogen…
Metabolism of estradiol-17&bgr; to 2-hydroxyestradiol, 4-hydroxyestradiol, 2-methoxyestradiol, and 4-methoxyestradiol contributes… 
Highly Cited
2006
Highly Cited
2006
In SHR aorta, calcium ionophore A-23187 releases prostacyclin and thromboxane A2 as endothelium-derived contracting factors.
In mature spontaneously hypertensive rats (SHR) and Wistar-Kyoto rats (WKY), acetylcholine and the calcium ionophore A-23187… 
2005
2005
Oxygenation of polyunsaturated long chain fatty acids by recombinant CYP4F8 and CYP4F12 and catalytic importance of Tyr-125 and Gly-328 of CYP4F8.
Highly Cited
2000
Highly Cited
2000
Identification of CYP4F8 in Human Seminal Vesicles as a Prominent 19-Hydroxylase of Prostaglandin Endoperoxides*
A novel cytochrome P450, CYP4F8, was recently cloned from human seminal vesicles. CYP4F8 was expressed in yeast. Recombinant… 
1986
1986
Prostacyclin and steroidogenesis in goat ovarian cell types in vitro.
1984
1984
The effect of prostaglandin synthesis inhibition on the direct stimulation of renin release from rabbit renal cortical slices.
Highly Cited
1982
Highly Cited
1982
Synergistic activation by collagen and 15-hydroxy-9 alpha,11 alpha-peroxidoprosta-5,13-dienoic acid (PGH2) of phosphatidylinositol metabolism and arachidonic acid release in human platelets.
Collagen stimulates the activation of phosphatidylinositol (PI)-specific phospholipase C (EC 3.1.4.10) in human platelets, as… 
1981
1981
Modification of human platelet response to sodium arachidonate by membrane modulation.
1977
1977
Thromboxane synthetase inhibitors as pharmacological tools: differential biochemical and biological effects on platelet suspensions.
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