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SNX 111

Known as: SNX-111 
 
National Institutes of Health

Papers overview

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Highly Cited
2003
Highly Cited
2003
In renal failure, parathyroid hormone (PTH) is not only involved in the genesis of disturbed calcium/phosphate metabolism and… Expand
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Highly Cited
1999
Highly Cited
1999
BACKGROUND AND PURPOSE Transient forebrain ischemia results in a 24- to 72-hour delayed loss of CA1 neurons. Previous work has… Expand
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1998
1998
We recently demonstrated that posttraumatic administration of the N-type calcium channel blocker SNX-111 (S) and a novel blood… Expand
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1997
1997
OBJECTIVE The objective was to assess the analgesic, antihyperesthesic, and anti-allodynic properties of SNX-111 in neuropathic… Expand
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Highly Cited
1997
Highly Cited
1997
We have recently demonstrated in a rat model that traumatic brain injury induces perturbation of cellular calcium homeostasis… Expand
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1997
1997
Cytosolic Ca2+ overload has been proposed as a main cause of neuronal injury during cerebral ischemia. SNX-111, a synthetic… Expand
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Highly Cited
1996
Highly Cited
1996
Male Sprague-Dawley rats were used to evaluate the antinociceptive properties of the selective N-type voltage-sensitive calcium… Expand
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1996
1996
Diffusion-weighted magnetic resonance imaging (DWI) is capable of noninvasively imaging acute cerebral ischemia. We demonstrate… Expand
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1994
1994
Neuronal voltage-sensitive calcium channels (VSCCs) are a diverse family of proteins that regulate entry of Ca2+ into neurons… Expand
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Highly Cited
1994
Highly Cited
1994
The role of spinal voltage-sensitive calcium channels (VSCC) in a behavioral model of prolonged nociception was examined in rats… Expand
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