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Plaque, Amyloid

Known as: Deposit, Amyloid, Deposits, Amyloid, Amyloid Deposits 
Accumulations of extracellularly deposited AMYLOID FIBRILS within tissues.
National Institutes of Health

Papers overview

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Highly Cited
2006
Highly Cited
2006
Mutations in the genes for amyloid precursor protein (APP) and presenilins (PS1, PS2) increase production of β-amyloid 42 (Aβ42… Expand
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Highly Cited
2005
Highly Cited
2005
Alzheimer's disease (AD) involves amyloid β (Aβ) accumulation, oxidative damage, and inflammation, and risk is reduced with… Expand
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Review
2004
Review
2004
SummaryEighty-three brains obtained at autopsy from nondemented and demented individuals were examined for extracellular amyloid… Expand
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Highly Cited
2004
Highly Cited
2004
This report describes the first human study of a novel amyloid‐imaging positron emission tomography (PET) tracer, termed… Expand
Highly Cited
2000
Highly Cited
2000
Amyloid plaques are a neuropathological hallmark of Alzheimer's disease (AD), but their relationship to neurodegeneration and… Expand
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Highly Cited
1999
Highly Cited
1999
Amyloid-β peptide (Aβ) seems to have a central role in the neuropathology of Alzheimer's disease (AD). Familial forms of the… Expand
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Highly Cited
1996
Highly Cited
1996
Transgenic mice overexpressing the 695-amino acid isoform of human Alzheimer β-amyloid (Aβ) precursor protein containing a Lys670… Expand
Highly Cited
1995
Highly Cited
1995
ALZHEIMER'S disease (AD) is the most common cause of progressive intellectual failure in aged humans. AD brains contain numerous… Expand
Highly Cited
1991
Highly Cited
1991
During the course of our immunohistochemical studies on the change of lipids in Alzheimer's disease brains by using antibody to… Expand
Highly Cited
1987
Highly Cited
1987
Alzheimer's disease1 is characterized by a widespread functional disturbance of the human brain. Fibrillar amyloid proteins are… Expand