ONO 1714

Known as: ONO-1714 
 
National Institutes of Health

Topic mentions per year

Topic mentions per year

2000-2012
024620002012

Papers overview

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2007
2007
It is generally assumed that inflammation influences carcinogenesis. We previously reported that dextran sodium sulfate (DSS… (More)
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2007
2007
STUDY OBJECTIVES High-concentration oxygen therapy is used to treat tissue hypoxia, but hyperoxia causes lung injury… (More)
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2006
2006
The expression of inducible nitric oxide synthase (iNOS) is markedly elevated in rat colon cancers induced by azoxymethane (AOM… (More)
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2004
2004
The overproduction of nitric oxide (NO) by inducible nitric oxide synthase (iNOS) may contribute to the pathophysiology of… (More)
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2003
2003
UNLABELLED Overproduction of nitric oxide by inducible nitric oxide synthase (iNOS) expressed in the lung is thought to play a… (More)
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2003
2003
BACKGROUND It is still controversial whether a major mode of cell death during hepatic ischemia-reperfusion (I/R) injuries is… (More)
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2002
2002
BACKGROUND Recently, a novel inhibitor of inducible nitric oxide synthase, ONO-1714, was developed. We evaluated the effect of… (More)
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2001
2001
OBJECTIVES Acute experimental pancreatitis (induced by cerulein) recently has been reported to cause marked diaphragmatic… (More)
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2001
2001
In mice with acute dextran sulfate sodium colitis, we examined the effect of inducible nitric oxide synthase inhibition by (1S,5S… (More)
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2000
2000
(1S,5S,6R,7R)-7-Chloro-3-imino-5-methyl-2-azabicyclo[4.1.0]heptane hydrochloride (ONO-1714), a novel cyclic amidine analogue… (More)
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