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Nitric Oxide Synthase Type III
Known as:
Nitric Oxide Synthase Type III [Chemical/Ingredient]
, ECNOS Enzyme
, ENOS Enzyme
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A CALCIUM-dependent, constitutively-expressed form of nitric oxide synthase found primarily in ENDOTHELIAL CELLS.
National Institutes of Health
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11 relations
In Blood
NANOS3 gene
NOS1 protein, human
NOS3 gene
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NOS3 protein, human
Papers overview
Semantic Scholar uses AI to extract papers important to this topic.
Review
2004
Review
2004
Pulmonary–Hepatic vascular Disorders (PHD)
R. Rodríguez-Roisín
,
M. Krowka
,
P. Hervé
,
M. Fallon
European Respiratory Journal
2004
Corpus ID: 15430962
⇓“The tantalizing problem of the connective link in cirrhotic patients between oxygen unsaturation and possible arteriovenous…
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Highly Cited
2003
Highly Cited
2003
Essential role of endothelial nitric oxide synthase for mobilization of stem and progenitor cells
A. Aicher
,
C. Heeschen
,
+5 authors
S. Dimmeler
Nature Network Boston
2003
Corpus ID: 1638889
Endothelial nitric oxide synthase (eNOS) is essential for neovascularization. Here we show that the impaired neovascularization…
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Highly Cited
2002
Highly Cited
2002
Acute cardiovascular protective effects of corticosteroids are mediated by non-transcriptional activation of endothelial nitric oxide synthase
A. Hafezi-Moghadam
,
T. Simoncini
,
+12 authors
J. Liao
Nature Network Boston
2002
Corpus ID: 13989981
Corticosteroids have been shown to exert beneficial effects in the treatment of acute myocardial infarction, but the precise…
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Highly Cited
2001
Highly Cited
2001
Hyperglycemia inhibits endothelial nitric oxide synthase activity by posttranslational modification at the Akt site.
Xue-liang Du
,
D. Edelstein
,
S. Dimmeler
,
Qida Ju
,
Chengyu Sui
,
M. Brownlee
Journal of Clinical Investigation
2001
Corpus ID: 16985773
Endothelial nitric oxide synthase (eNOS) is activated by phosphorylation of serine 1177 by the protein kinase Akt/PKB. Since…
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Highly Cited
2000
Highly Cited
2000
Regulation of endothelial constitutive nitric oxide synthase gene expression in endothelial cells and in vivo : a specific vascular action of insulin.
K. Kuboki
,
Zhen Y Jiang
,
+7 authors
G. King
Circulation
2000
Corpus ID: 9302484
BACKGROUND The vasodilatory effect of insulin can be acute or increase with time from 1 to 7 hours, suggesting that insulin may…
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Highly Cited
2000
Highly Cited
2000
Membrane Estrogen Receptor Engagement Activates Endothelial Nitric Oxide Synthase via the PI3-Kinase–Akt Pathway in Human Endothelial Cells
M. Haynes
,
Diviya Sinha
,
+5 authors
J. Bender
Circulation Research
2000
Corpus ID: 8277073
17&bgr;-Estradiol (E2) is a rapid activator of endothelial nitric oxide synthase (eNOS). The product of this activation event, NO…
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Highly Cited
2000
Highly Cited
2000
In vivo delivery of the caveolin-1 scaffolding domain inhibits nitric oxide synthesis and reduces inflammation
M. Bucci
,
J. Gratton
,
+4 authors
W. Sessa
Nature Network Boston
2000
Corpus ID: 30152502
Caveolin-1, the primary coat protein of caveolae, has been implicated as a regulator of signal transduction through binding of…
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Highly Cited
1999
Highly Cited
1999
Regulation of endothelium-derived nitric oxide production by the protein kinase Akt
D. Fulton
,
J. Gratton
,
+6 authors
W. Sessa
Nature
1999
Corpus ID: 4394717
Endothelial nitric oxide synthase (eNOS) is the nitric oxide synthase isoform responsible for maintaining systemic blood pressure…
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Highly Cited
1998
Highly Cited
1998
Superoxide generation by endothelial nitric oxide synthase: the influence of cofactors.
J. Vasquez-Vivar
,
B. Kalyanaraman
,
+5 authors
K. Pritchard
Proceedings of the National Academy of Sciences…
1998
Corpus ID: 665840
The mechanism of superoxide generation by endothelial nitric oxide synthase (eNOS) was investigated by the electron spin…
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Highly Cited
1997
Highly Cited
1997
High glucose increases nitric oxide synthase expression and superoxide anion generation in human aortic endothelial cells.
F. Cosentino
,
K. Hishikawa
,
Z. Katusic
,
T. Lüscher
Circulation
1997
Corpus ID: 24568462
BACKGROUND Hyperglycemia is a primary cause of premature vascular disease. Endothelial cell dysfunction characterized by…
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