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NOD.CB17-Prkdc-scid/J Mouse

Known as: NOD.SCID, NOD/LtSz-Prkdc-scid/J, NOD SCID 
Originally derived by Prochazka et al (1992) at Jackson Laboratories, by crossing a C.B-17 congenic background mouse with the scid mutation to a… Expand
National Institutes of Health

Papers overview

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Highly Cited
2016
Highly Cited
2016
Uncoupling protein 1 (UCP1) is highly expressed in brown adipose tissue, where it generates heat by uncoupling electron transport… Expand
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Highly Cited
2011
Highly Cited
2011
Rationale: Transgenic Notch reporter mice express enhanced green fluorescent protein in cells with C-promoter binding factor-1… Expand
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Highly Cited
2010
Highly Cited
2010
"Humanized" mouse models created by engraftment of immunodeficient mice with human hematolymphoid cells or tissues are an… Expand
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Review
2008
Review
2008
Mesenchymal stem cells (MSCs) are pluripotent stromal cells that have the potential to give rise to cells of diverse lineages… Expand
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Highly Cited
2005
Highly Cited
2005
Here we report that a new nonobese diabetic/severe combined immunodeficient (NOD/SCID) mouse line harboring a complete null… Expand
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Highly Cited
2005
Highly Cited
2005
Internal tandem duplication mutations of the FLT3 gene (FLT3/ITD mutations) are the most frequent molecular abnormality in acute… Expand
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Highly Cited
2003
Highly Cited
2003
Vascular endothelial growth factor (VEGF) and its receptors (VEGFR) have been implicated in promoting solid tumor growth and… Expand
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Highly Cited
2003
Highly Cited
2003
Infection with Schistosoma mansoni (S. mansoni) or exposure to eggs from this helminth inhibits the development of type 1… Expand
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Highly Cited
2001
Highly Cited
2001
An increasing number of studies have documented the central role of T cell costimulation in autoimmunity. Here we show that the… Expand
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Highly Cited
1995
Highly Cited
1995
The scid mutation was backcrossed ten generations onto the NOD/Lt strain background, resulting in an immunodeficient stock (NOD… Expand
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