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NOD.CB17-Prkdc-scid/J Mouse

Known as: NOD.SCID, NOD/LtSz-Prkdc-scid/J, NOD SCID 
Originally derived by Prochazka et al (1992) at Jackson Laboratories, by crossing a C.B-17 congenic background mouse with the scid mutation to a… 
National Institutes of Health

Papers overview

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Highly Cited
2016
Highly Cited
2016
Uncoupling protein 1 (UCP1) is highly expressed in brown adipose tissue, where it generates heat by uncoupling electron transport… 
Highly Cited
2011
Highly Cited
2011
Rationale: Transgenic Notch reporter mice express enhanced green fluorescent protein in cells with C-promoter binding factor-1… 
Review
2008
Review
2008
Mesenchymal stem cells (MSCs) are pluripotent stromal cells that have the potential to give rise to cells of diverse lineages… 
Highly Cited
2005
Highly Cited
2005
Internal tandem duplication mutations of the FLT3 gene (FLT3/ITD mutations) are the most frequent molecular abnormality in acute… 
Highly Cited
2003
Highly Cited
2003
Infection with Schistosoma mansoni (S. mansoni) or exposure to eggs from this helminth inhibits the development of type 1… 
Highly Cited
2003
Highly Cited
2003
Vascular endothelial growth factor (VEGF) and its receptors (VEGFR) have been implicated in promoting solid tumor growth and… 
Highly Cited
2002
Highly Cited
2002
c null mice and NOD/Shi-scid mice. When human CD34 cells from umbilical cord blood were transplanted into this strain, the… 
Highly Cited
2001
Highly Cited
2001
An increasing number of studies have documented the central role of T cell costimulation in autoimmunity. Here we show that the… 
Highly Cited
1995
Highly Cited
1995
The scid mutation was backcrossed ten generations onto the NOD/Lt strain background, resulting in an immunodeficient stock (NOD…