Skip to search form
Skip to main content
Skip to account menu
Semantic Scholar
Semantic Scholar's Logo
Search 217,273,823 papers from all fields of science
Search
Sign In
Create Free Account
Murid herpesvirus 1
Known as:
Cytomegalovirus, Murine
, Herpesvirus 1 (beta), Murid
, Mouse Cytomegaloviruses
Expand
The type species of MUROMEGALOVIRUS infecting mice.
National Institutes of Health
Create Alert
Alert
Related topics
Related topics
2 relations
Betaherpesvirinae
Broader (1)
Muromegalovirus
Papers overview
Semantic Scholar uses AI to extract papers important to this topic.
Review
2019
Review
2019
The molecular machinery of regulated cell death
D. Tang
,
R. Kang
,
T. Berghe
,
P. Vandenabeele
,
G. Kroemer
Cell Research
2019
Corpus ID: 96435429
Cells may die from accidental cell death (ACD) or regulated cell death (RCD). ACD is a biologically uncontrolled process, whereas…
Expand
Highly Cited
2012
Highly Cited
2012
DAI/ZBP1/DLM-1 complexes with RIP3 to mediate virus-induced programmed necrosis that is targeted by murine cytomegalovirus vIRA.
J. Upton
,
W. Kaiser
,
E. Mocarski
Cell Host and Microbe
2012
Corpus ID: 5340413
Highly Cited
2012
Highly Cited
2012
Cytomegalovirus reactivation after allogeneic transplantation promotes a lasting increase in educated NKG2C+ natural killer cells with potent function.
B. Foley
,
S. Cooley
,
+7 authors
Jeffrey S. Miller
Blood
2012
Corpus ID: 206906322
During mouse cytomegalovirus (CMV) infection, a population of Ly49H(+) natural killer (NK) cells expands and is responsible for…
Expand
Highly Cited
2011
Highly Cited
2011
RIP3 mediates the embryonic lethality of caspase-8-deficient mice
W. Kaiser
,
J. Upton
,
+5 authors
E. Mocarski
Nature
2011
Corpus ID: 4372260
Apoptosis and necroptosis are complementary pathways controlled by common signalling adaptors, kinases and proteases; among these…
Expand
Highly Cited
2007
Highly Cited
2007
Herpesvirus latency confers symbiotic protection from bacterial infection
E. Barton
,
D. White
,
+5 authors
H. Virgin
Nature
2007
Corpus ID: 4425405
All humans become infected with multiple herpesviruses during childhood. After clearance of acute infection, herpesviruses enter…
Expand
Highly Cited
2006
Highly Cited
2006
The Unc93b1 mutation 3d disrupts exogenous antigen presentation and signaling via Toll-like receptors 3, 7 and 9
K. Tabeta
,
K. Hoebe
,
+16 authors
B. Beutler
Nature Immunology
2006
Corpus ID: 33401155
Here we have identified 'triple D' (3d), a recessive N-ethyl-N-nitrosourea-induced mutation and phenotype in which no signaling…
Expand
Highly Cited
2004
Highly Cited
2004
Toll-like receptors 9 and 3 as essential components of innate immune defense against mouse cytomegalovirus infection.
K. Tabeta
,
P. Georgel
,
+10 authors
B. Beutler
Proceedings of the National Academy of Sciences…
2004
Corpus ID: 40268565
Several subsets of dendritic cells have been shown to produce type I IFN in response to viral infections, thereby assisting the…
Expand
Highly Cited
2003
Highly Cited
2003
Identification of Lps2 as a key transducer of MyD88-independent TIR signalling
K. Hoebe
,
Xin Du
,
+11 authors
B. Beutler
Nature
2003
Corpus ID: 15608748
In humans, ten Toll-like receptor (TLR) paralogues sense molecular components of microbes, initiating the production of cytokine…
Expand
Review
2002
Review
2002
Role of chemokines in the biology of natural killer cells
M. Robertson
Journal of Leukocyte Biology
2002
Corpus ID: 720060
Natural killer (NK) cells participate in innate and adaptive immuneresponses to obligate intracellular pathogens and malignant…
Expand
Highly Cited
1996
Highly Cited
1996
Analysis of the complete DNA sequence of murine cytomegalovirus
W. Rawlinson
,
H. Farrell
,
B. Barrell
Journal of Virology
1996
Corpus ID: 9465745
The complete DNA sequence of the Smith strain of murine cytomegalovirus (MCMV) was determined from virion DNA by using a whole…
Expand
By clicking accept or continuing to use the site, you agree to the terms outlined in our
Privacy Policy
(opens in a new tab)
,
Terms of Service
(opens in a new tab)
, and
Dataset License
(opens in a new tab)
ACCEPT & CONTINUE