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MHY 218

Known as: MHY-218, MHY218 
National Institutes of Health

Papers overview

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Review
2017
Review
2017
A systematic review of hybrid brain-computer interfaces: Taxonomy and usability perspectives
A new Brain-Computer Interface (BCI) technique, which is called a hybrid BCI, has recently been proposed to address the… 
2016
2016
Increased therapeutic efficacy of a newly synthesized tyrosinase inhibitor by solid lipid nanoparticles in the topical treatment of hyperpigmentation
Hyperpigmentation caused by melanin overproduction is a major skin disorder in humans. Inhibition of tyrosinase, a key regulator… 
2015
2015
MHY218-induced apoptotic cell death is enhanced by the inhibition of autophagy in AGS human gastric cancer cells.
We previously reported the anticancer effects of MHY218, which is a hydroxamic acid derivative, in HCT116 human colon cancer… 
2014
2014
A novel hydroxamic acid derivative, MHY218, induces apoptosis and cell cycle arrest through downregulation of NF-κB in HCT116 human colon cancer cells.
Colorectal cancer (CRC) is one of the most common malignant diseases and frequent cause of cancer deaths in the world. In spite… 
2011
2011
A new synthetic HDAC inhibitor, MHY218, induces apoptosis or autophagy-related cell death in tamoxifen-resistant MCF-7 breast cancer cells
SummaryAcquired resistance to tamoxifen (Tam) is a critical problem in breast cancer therapy. Therefore, new potential strategies… 
2011
2011
Abstract 182: Novel hydroxamic acid-derivative, MHY218, induces apoptosis and cell cycle arrest in HCT116 human colon cancer cells
In this study, we investigated the anti-proliferative and pro-apoptotic properties of novel hydroxamic acid-derivative, MHY218… 
2010
2010
Anticancer effects of the MHY218 novel hydroxamic acid-derived histone deacetylase inhibitor in human ovarian cancer cells.
To investigate the anticancer effects of the novel hydroxamic acid-derived histone deacetylase (HDAC) inhibitor MHY218, its… 
2009
2009
A NOVEL HYDROXAMIC ACID-DERIVED HISTONE DEACETYLASE INHIBITOR, MHY-218, INDUCES AUTOPHAGY-DEPENDENT CELL DEATH IN TAMOXIFEN-RESISTANCE MCF-7 CELLS
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