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M40401

Known as: M 40401 
 
National Institutes of Health

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1 relation

Broader (1)

Organometallic Compounds

Papers overview

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2014
2014
QKI deficiency promotes FoxO1 mediated nitrosative stress and endoplasmic reticulum stress contributing to increased vulnerability to ischemic injury in diabetic heart.
Hearts of diabetic individuals are susceptible to ischemia/reperfusion (I/R) injury. The RNA-binding protein Quaking (QKI) is… Expand
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Highly Cited
2010
Highly Cited
2010
Oxidative stress regulates left ventricular PDE5 expression in the failing heart.
BACKGROUND Phosphodiesterase type 5 (PDE5) inhibition has been shown to exert profound beneficial effects in the failing heart… Expand
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2005
2005
Increased superoxide production causes coronary endothelial dysfunction and depressed oxygen consumption in the failing heart.
This study examined whether increased superoxide (O(2)(-).) production contributes to coronary endothelial dysfunction and… Expand
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2004
2004
Reduction in the development of cerulein-induced acute pancreatitis by treatment with M40401, a new selective superoxide dismutase mimetic.
Oxidative stress plays an important role in the early stage of acute pancreatitis, as well as in the associated multiple organ… Expand
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2004
2004
The protective effect of superoxide dismutase mimetic M40401 on balloon injury-related neointima formation: role of the lectin-like oxidized low-density lipoprotein receptor-1.
Lectin-like oxidized low-density lipoprotein receptor-1 (LOX-1), the principal receptor for oxidized low-density lipoprotein (ox… Expand
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2004
2004
Superoxide dismutase mimetic m40401 reduces ischemia-reperfusion injury and graft coronary artery disease in rodent cardiac allografts.
BACKGROUND The oxidative stress associated with ischemia-reperfusion (I/R) of cardiac allografts leads to production of injurious… Expand
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2003
2003
Modulation of serum cytokine levels by a novel superoxide dismutase mimetic, M40401, in an Escherichia coli model of septic shock: correlation with preserved circulating catecholamines.
OBJECTIVES We have shown previously that inactivation of catecholamines by superoxide anions contributes to the loss of vascular… Expand
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Highly Cited
2003
Highly Cited
2003
Diazoxide induces delayed pre-conditioning in cultured rat cortical neurons.
We investigated the effect of diazoxide on neuronal survival in primary cultures of rat cortical neurons against oxygen-glucose… Expand
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2003
2003
Diazoxide pretreatment induces delayed preconditioning in astrocytes against oxygen glucose deprivation and hydrogen peroxide-induced toxicity.
Recent studies suggest that activation of mitochondrial ATP-sensitive potassium channels (mK(ATP)) with diazoxide can protect… Expand
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Highly Cited
2001
Highly Cited
2001
Protective effects of a new stable, highly active SOD mimetic, M40401 in splanchnic artery occlusion and reperfusion.
1. Splanchnic artery occlusion shock (SAO) causes an enhanced formation of reactive oxygen species (ROS), which contribute to the… Expand
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