LY 231617

Known as: LY-231617, LY231617 
 
National Institutes of Health

Papers overview

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2005
2005
Activation of transcription factor nuclear factor-κB (NF-κB) can result in enhanced de novo synthesis of both proteins that… (More)
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2003
2003
The role of reactive oxygen species in the pathogenesis of the neurotoxicity associated with ischemia-reperfusion, was… (More)
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1998
1998
Lipid peroxidation is a major consequence of oxidative stress and an important cause of neuronal damage in ischaemic injuries and… (More)
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1997
1997
Recent studies have shown that the novel antioxidant LY231617 protects against ischaemia-induced neuronal damage in rat models of… (More)
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1997
1997
Global cerebral ischemia leads to selective neuronal damage in the CA1 sector of the hippocampus and in the striatum. This… (More)
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Highly Cited
1996
Highly Cited
1996
BACKGROUND Reactive oxygen species may mediate tissue injury in inflammatory bowel disease. Aminosalicylates have antioxidant… (More)
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1996
1996
The antioxidant LY231617 has previously been shown to offer significant protection against postischemic cell death in the… (More)
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1995
1995
In this study the effect of LY 231617, an antioxidant, on spatial learning deficit and on neuronal damage following transient… (More)
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1993
1993
BACKGROUND AND PURPOSE In the rat four-vessel occlusion model with 30 minutes of ischemia most agents have failed to be of… (More)
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