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KU 60019

Known as: KU-60019, KU60019 
 
National Institutes of Health

Papers overview

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2019
2019
BackgroundMYCN amplification or N-Myc overexpression is found in approximately 40% NEPC and up to 20% CRPC patients. N-Myc has… Expand
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2019
2019
Despite advances in glioblastoma (GBM) therapy, prognosis of the disease remains poor with a low survival rate. Cannabidiol (CBD… Expand
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2015
2015
We have recently reported that glioblastoma (GB)‐initiating cells (GIC) with low expression and/or mutation of TP53 and high… Expand
2015
2015
Ataxia telangiectasia mutated (ATM) is an important signaling molecule in the DNA damage response (DDR). ATM loss of function can… Expand
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2014
2014
We have previously shown that pharmacological inhibition of ataxia telangiectasia mutated (ATM) protein sensitizes glioblastoma… Expand
2014
2014
It has previously been reported that KU60019, as a highly effective radiosensitizer, inhibits the DNA damage response and blocks… Expand
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Highly Cited
2013
Highly Cited
2013
Purpose: Glioblastoma multiforme (GBM) is the most lethal form of brain cancer with a median survival of only 12 to 15 months… Expand
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Highly Cited
2012
Highly Cited
2012
Glioblastoma multiforme (GBM) is notoriously resistant to treatment. Therefore, new treatment strategies are urgently needed. ATM… Expand
2011
2011
Dear Editor, The purpose of this letter is to address the activity of CGK733, a small molecule that was reported to inhibit… Expand
Highly Cited
2009
Highly Cited
2009
Ataxia telangiectasia (A-T) mutated (ATM) is critical for cell cycle checkpoints and DNA repair. Thus, specific small molecule… Expand
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