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HMJ-38

 
National Institutes of Health

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2014
2014
Cell death caused by quinazolinone HMJ-38 challenge in oral carcinoma CAL 27 cells: dissections of endoplasmic reticulum stress, mitochondrial dysfunction and tumor xenografts.
BACKGROUND This investigation clearly clarified the synthesized and antimitotic compound, 2-(3'-methoxyphenyl)-6-pyrrolidinyl-4… Expand
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2014
2014
Effect of DNA damage response by quinazolinone analogue HMJ-38 on human umbilical vein endothelial cells: evidence for γH2A.X and DNA-PK-dependent pathway.
The present study aims to explore the mechanism of quinazolinone analogue HMJ-38-induced DNA damage in endothelial cells in vitro… Expand
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2013
2013
Newly synthesized quinazolinone HMJ-38 suppresses angiogenetic responses and triggers human umbilical vein endothelial cell apoptosis through p53-modulated Fas/death receptor signaling.
The current study aims to investigate the antiangiogenic responses and apoptotic death of human umbilical vein endothelial cells… Expand
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2004
2004
Selective induction of G2/M arrest and apoptosis in HL-60 by a potent anticancer agent, HMJ-38.
We previously reported that HMJ-38 was the most potent 2-phenyl-4-quinozolinone derivative in inhibiting tubulin polymerization… Expand
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1993
1993
2-(3''-Methoxyphenyl)-6-pyrrolidinyl-4-quinazolinone (HMJ-38) 對HL-60血癌細胞及; Studies on the Anticancer Mechanisms of 2-(3''-Methoxyphenyl)-6-pyrrolidinyl-4-quinazolinone (HMJ-38) in HL-60 cells and in…
 
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