FANCI GENE

Known as: Fanconi Anemia, Complementation Group I Gene, FLJ10719, FANCI 
This gene plays a role in the mediation of DNA repair.
National Institutes of Health

Papers overview

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2013
2013
Fanconi Anemia (FA) and Bloom Syndrome share overlapping phenotypes including spontaneous chromosomal abnormalities and increased… (More)
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2012
2012
FANCI and FANCD2 form a complex, and play essential roles in the repair of interstrand DNA crosslinks (ICLs) by the Fanconi… (More)
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Highly Cited
2011
Highly Cited
2011
Various cyclin-dependent kinase (Cdk) complexes have been implicated in the regulation of transcription. In this study, we… (More)
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Highly Cited
2011
Highly Cited
2011
Fanconi anemia is a cancer predisposition syndrome caused by defects in the repair of DNA interstrand cross-links (ICLs). Central… (More)
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Highly Cited
2010
Highly Cited
2010
Fanconi anemia (FA) is caused by mutations in 13 Fanc genes and renders cells hypersensitive to DNA interstrand cross-linking… (More)
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Highly Cited
2008
Highly Cited
2008
In response to DNA damage or replication fork stress, the Fanconi anemia pathway is activated, leading to monoubiquitination of… (More)
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Highly Cited
2008
Highly Cited
2008
A key step in the Fanconi anemia (FA) tumor suppressor pathway is the site-specific monoubiquitination of the FANCD2 protein… (More)
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Highly Cited
2007
Highly Cited
2007
Fanconi anemia (FA) is a developmental and cancer-predisposition syndrome caused by mutations in genes controlling DNA… (More)
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Highly Cited
2007
Highly Cited
2007
Activation of the Fanconi anemia (FA) DNA damage–response pathway results in the monoubiquitination of FANCD2, which is regulated… (More)
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Highly Cited
2007
Highly Cited
2007
To identify the gene underlying Fanconi anemia (FA) complementation group I we studied informative FA-I families by a genome-wide… (More)
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