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FAM49A gene
Known as:
FLJ11080
, family with sequence similarity 49 member A
, FAM49A
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National Institutes of Health
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Related topics
Related topics
1 relation
FRAXF gene
Papers overview
Semantic Scholar uses AI to extract papers important to this topic.
2019
2019
Exploring GRHL3 polymorphisms and SNP-SNP interactions in the risk of nonsyndromic oral clefts in the Brazilian population.
Camilla de Marchi Sanches Azevedo
,
R. Machado
,
+4 authors
A. L. Rangel
Oral Diseases
2019
Corpus ID: 203609236
OBJECTIVE To investigate the association of single-nucleotide polymorphisms (SNP) in grainyhead-like-3 (GRHL3) and to verify its…
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2018
2018
Identification of novel lncRNAs regulated by the TAL1 complex in T-cell acute lymphoblastic leukemia
P. C. T. Ngoc
,
S. Tan
,
+5 authors
T. Sanda
Leukemia
2018
Corpus ID: 4830549
TAL1/SCL is one of the most prevalent oncogenes in T-cell acute lymphoblastic leukemia (T-ALL). TAL1 and its regulatory partners…
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2018
2018
Genetic variants of MGMT, RHPN2, and FAM49A contributed to susceptibility of nonsyndromic orofacial clefts in a Chinese population
Chunyu Chen
,
Qiang Guo
,
+6 authors
Tao Song
Journal of Oral Pathology & Medicine
2018
Corpus ID: 49428200
BACKGROUND The role of underlying genetic factors in the pathogenesis of nonsyndromic orofacial clefts (NSOC) remains poorly…
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Highly Cited
2016
Highly Cited
2016
A multi-ethnic genome-wide association study identifies novel loci for non-syndromic cleft lip with or without cleft palate on 2p24.2, 17q23 and 19q13.
E. Leslie
,
J. Carlson
,
+52 authors
M. Marazita
Human Molecular Genetics
2016
Corpus ID: 27662362
Orofacial clefts (OFCs), which include non-syndromic cleft lip with or without cleft palate (CL/P), are among the most common…
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Highly Cited
2010
Highly Cited
2010
Gene amplification as double minutes or homogeneously staining regions in solid tumors: origin and structure.
C. Storlazzi
,
A. Lonoce
,
+13 authors
M. Rocchi
Genome Research
2010
Corpus ID: 13308442
Double minutes (dmin) and homogeneously staining regions (hsr) are the cytogenetic hallmarks of genomic amplification in cancer…
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