Skip to search form
Skip to main content
Skip to account menu
Semantic Scholar
Semantic Scholar's Logo
Search 205,587,809 papers from all fields of science
Search
Sign In
Create Free Account
DRAIC gene
Known as:
downregulated RNA in androgen independent cells
, LONG NONCODING RNA DRAIC
, DOWNREGULATED RNA IN ANDROGEN-INDEPENDENT CELLS, NONCODING
Expand
National Institutes of Health
Create Alert
Alert
Papers overview
Semantic Scholar uses AI to extract papers important to this topic.
2020
2020
Long noncoding RNA DRAIC inhibits prostate cancer progression by interacting with IKK to inhibit NF-κB activation.
S. Saha
,
M. Kiran
,
+4 authors
Anindya Dutta
Cancer research
2020
Corpus ID: 209751429
DRAIC is a 1.7 kb spliced long noncoding RNA downregulated in castration-resistant advanced prostate cancer. Decreased DRAIC…
Expand
2020
2020
LncRNA DRAIC inhibits proliferation and metastasis of gastric cancer cells through interfering with NFRKB deubiquitination mediated by UCHL5
Zheng Zhang
,
Xiaoxuan Hu
,
Jia Kuang
,
Jinmao Liao
,
Q. Yuan
Cellular & Molecular Biology Letters
2020
Corpus ID: 216128676
Background Long non-coding RNA (lncRNA) as a widespread and pivotal epigenetic molecule participates in the occurrence and…
Expand
2020
2020
Regulatory effect of LncRNA DRAIC/miR-149-5p/NFIB molecular network on autophagy of esophageal cancer cells and its biological behavior.
Furong Li
,
Xinghua Zhou
,
Ming Chen
,
W. Fan
Experimental and molecular pathology
2020
Corpus ID: 220522081
2020
2020
Re: Long Noncoding RNA DRAIC Inhibits Prostate Cancer Progression by Interacting with IKK to Inhibit NF-κB Activation.
A. Atala
The Journal of urology
2020
Corpus ID: 219909369
available at http://pubmed.ncbi.nlm.nih.gov/31900260 Editorial Comment: An androgen repressed cytoplasmic long noncoding (lnc…
Expand
2019
2019
A high-throughput screen identifies the long non-coding RNA DRAIC as a regulator of autophagy
Imke Tiessen
,
Marie H Abildgaard
,
+6 authors
A. Lund
Oncogene
2019
Corpus ID: 77395286
Autophagy is a conserved degradation process that occurs in all eukaryotic cells and its dysfunction has been associated with…
Expand
2019
2019
Long noncoding RNA DRAIC acts as a microRNA-122 sponge to facilitate nasopharyngeal carcinoma cell proliferation, migration and invasion via regulating SATB1
B. Liao
,
Zhi Wang
,
Yaqiong Zhu
,
Meiqun Wang
,
Yuehui Liu
Artificial cells, nanomedicine, and biotechnology
2019
Corpus ID: 202003614
Abstract Increasing evidences have revealed that long noncoding RNAs (lncRNAs) are frequently involved in various cancers…
Expand
2018
2018
Upregulation of Long Non-Coding RNA DRAIC Correlates with Adverse Features of Breast Cancer
Dan Zhao
,
Jin-Tang Dong
Non-coding RNA
2018
Corpus ID: 56492364
DRAIC (also known as LOC145837 and RP11-279F6.1), is a long non-coding RNA associated with several types of cancer including…
Expand
Review
2017
Review
2017
Current Insights into Long Non-Coding RNAs (LncRNAs) in Prostate Cancer
M. Smolle
,
T. Bauernhofer
,
K. Pummer
,
G. Calin
,
M. Pichler
International journal of molecular sciences
2017
Corpus ID: 4791891
The importance of long non-coding RNAs (lncRNAs) in the pathogenesis of various malignancies has been uncovered over the last few…
Expand
2016
2016
Androgens induce a distinct response of epithelial-mesenchymal transition factors in human prostate cancer cells
J. Colditz
,
Benjamin Rupf
,
C. Maiwald
,
A. Baniahmad
Molecular and Cellular Biochemistry
2016
Corpus ID: 8342787
Inhibition of the androgen receptor (AR) is a major target of prostate cancer (PCa) therapy. However, prolonged androgen…
Expand
Highly Cited
2015
Highly Cited
2015
The lncRNA DRAIC/PCAT29 Locus Constitutes a Tumor-Suppressive Nexus
K. Sakurai
,
Brian J. Reon
,
Jordan Anaya
,
Anindya Dutta
Molecular Cancer Research
2015
Corpus ID: 20351246
Long noncoding RNAs (lncRNA) are emerging as major regulators of cellular phenotypes and implicated as oncogenes or tumor…
Expand
By clicking accept or continuing to use the site, you agree to the terms outlined in our
Privacy Policy
,
Terms of Service
, and
Dataset License
ACCEPT & CONTINUE