CPU0213

 
National Institutes of Health

Topic mentions per year

Topic mentions per year

2006-2016
024620062016

Papers overview

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2011
2011
OBJECTIVES Abnormal vascular activity in diabetes is related not only to impaired nitric oxide bioavailability but also to… (More)
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2009
2009
BACKGROUND/AIMS NADPH oxidase(NOX) is the main source of reactive oxygen species (ROS) in diabetic nephropathy (DN). Activation… (More)
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2009
2009
1. The aim of the present study was to explore the effects of CPU0213, a dual endothelin ET(A)/ET(B) receptor antagonist, and… (More)
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2008
2008
Downregulation of FKBP12.6 and sarcoplasmic reticulum Ca(2+) ATPase (SERCA2a) contributes to sudden cardiac death and heart… (More)
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2008
2008
An increase in reactive oxygen species (ROS) through NADPH oxidase activation frequently follows stress that activates beta… (More)
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2007
2007
Arrhythmias correlate with disorders of either K(2+) channels in sarcolemma or calcium modulating system in sarcoplasmic… (More)
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2007
2007
1. The aims of the present study were to examine whether: (i) upregulation of the endothelin (ET) pathway is involved in… (More)
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2007
2007
AbstractAim:The downregulation of phospholamban (PLB) and FKBP12.6 as a result of β-receptor activation is involved in the… (More)
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2006
2006
The depressed sarcoplasmic reticulum (SR) Ca2+-ATPase (SERCA2a) and Ca2+-release channels (ryanodine receptor RyR2) are involved… (More)
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2006
2006
AbstractAim:To examine whether a novel endothelin receptor antagonist, CPU0213, is effective in relieving the acute renal failure… (More)
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