Skip to search formSkip to main contentSkip to account menu

Androgen Receptor Splice Variant 3, Human

Known as: Androgen Receptor Splice Variant 7, Androgen Receptor Splice Variant 3, AR-v7 
Androgen receptor protein that is comprised of the amino acids encoded by exons 1-3 and those encoded by cryptic exon 3 (CE3) of the human AR gene… 
National Institutes of Health

Related topics

Related topics

21 relations
AR geneBiologic DevelopmentCell Differentiation processCell Nucleus

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
Highly Cited
2018
Highly Cited
2018
Assessment of the Validity of Nuclear-Localized Androgen Receptor Splice Variant 7 in Circulating Tumor Cells as a Predictive Biomarker for Castration-Resistant Prostate Cancer
Importance A blood test to determine whether to treat patients with metastatic castration-resistant prostate cancer (mCRPC) with… 
Highly Cited
2017
Highly Cited
2017
The Detection of Androgen Receptor Splice Variant 7 in Plasma-derived Exosomal RNA Strongly Predicts Resistance to Hormonal Therapy in Metastatic Prostate Cancer Patients.
Highly Cited
2016
Highly Cited
2016
Association of AR-V7 on Circulating Tumor Cells as a Treatment-Specific Biomarker With Outcomes and Survival in Castration-Resistant Prostate Cancer.
Importance A critical decision in the management of metastatic castration-resistant prostate cancer (mCRPC) is when to administer… 
Highly Cited
2015
Highly Cited
2015
Efficacy of Cabazitaxel in Castration-resistant Prostate Cancer Is Independent of the Presence of AR-V7 in Circulating Tumor Cells.
Highly Cited
2015
Highly Cited
2015
Androgen receptor splice variants contribute to prostate cancer aggressiveness through induction of EMT and expression of stem cell marker genes
The mechanism(s) by which androgen receptor (AR) splice variants contribute to castration‐resistant prostate cancer (CRPC) is… 
Highly Cited
2014
Highly Cited
2014
Mechanisms of the androgen receptor splicing in prostate cancer cells
Prostate tumors develop resistance to androgen deprivation therapy (ADT) by multiple mechanisms, one of which is to express… 
Review
2012
Review
2012
ASC-J9 suppresses castration-resistant prostate cancer growth through degradation of full-length and splice variant androgen receptors.
Early studies suggested androgen receptor (AR) splice variants might contribute to the progression of prostate cancer (PCa) into… 
Highly Cited
2010
Highly Cited
2010
Constitutively active androgen receptor splice variants expressed in castration-resistant prostate cancer require full-length androgen receptor
Androgen receptor (AR) splice variants lacking the ligand binding domain (ARVs), originally isolated from prostate cancer cell… 
Highly Cited
2009
Highly Cited
2009
A novel androgen receptor splice variant is up-regulated during prostate cancer progression and promotes androgen depletion-resistant growth.
The androgen receptor (AR) plays a key role in progression to incurable androgen ablation-resistant prostate cancer (PCA). We… 
Highly Cited
2004
Highly Cited
2004
Escherichia coli Glutamate- and Arginine-Dependent Acid Resistance Systems Increase Internal pH and Reverse Transmembrane Potential
ABSTRACT Due to the acidic nature of the stomach, enteric organisms must withstand extreme acid stress for colonization and… 
By clicking accept or continuing to use the site, you agree to the terms outlined in our Privacy Policy, Terms of Service, and Dataset License